A
Arvind Ingle
Researcher at Cancer Research Institute
Publications - 58
Citations - 1448
Arvind Ingle is an academic researcher from Cancer Research Institute. The author has contributed to research in topics: Cancer & Metastasis. The author has an hindex of 23, co-authored 49 publications receiving 1289 citations. Previous affiliations of Arvind Ingle include Tata Memorial Hospital & Bhabha Atomic Research Centre.
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Novel laboratory mouse papillomavirus (MusPV) infection.
TL;DR: The presence of a mouse PV, designated MusPV, was confirmed by amplification of PV DNA with degenerative primers specific for PVs by the first of a PV and its related disease in laboratory mice.
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Plakophilin3 downregulation leads to a decrease in cell adhesion and promotes metastasis
Samrat T. Kundu,Prajakta Gosavi,Nileema Khapare,Rachana Patel,Amol S. Hosing,Girish B. Maru,Arvind Ingle,James A. DeCaprio,Sorab N. Dalal +8 more
TL;DR: Data indicate that plakophilin3 loss leads to a decrease in cell–cell adhesion leading to the stimulation of neoplastic progression and metastasis.
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Implications of cytokeratin 8/18 filament formation in stratified epithelial cells: induction of transformed phenotype.
TL;DR: It appears that increased expression of CK8 in some way changes the phenotypic characteristics of stratified epithelial cells, resulting in malignant transformation.
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Chemopreventive efficacy of curcumin-free aqueous turmeric extract in 7,12-dimethylbenz[a]anthracene-induced rat mammary tumorigenesis
TL;DR: The present data clearly indicate that dietary administration of T/ETE showed strong chemopreventive activity during initiation as well as post-initiation phases of DMBA-induced rat mammary tumorigenesis while CFATE was found to be weakly active only when it was administered during the post-Initiation phase.
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Subchronic oral toxicity of turmeric and ethanolic turmeric extract in female mice and rats
TL;DR: Subchronic oral toxicity of turmeric and ethanolic turmeric extract was studied in female Swiss mice and Wistar rats and they were found to be more vulnerable to turmeric-induced hepatotoxicity than rats.