Barbara Wessner

TL;DR: The data provide strong evidence that 1,25(OH)2D3 primes monocytes to respond less effectively to bacterial cell wall components in a VDR‐dependent mechanism, most likely due to decreased levels of TLR2 and TLR4.

TL;DR: In the present special issue, several original and review articles addressing potential nutritional, pharmacological, and behavioral strategies that could be used to counteract inflammatory process associated with a variety of diseases including obesity, periodontal disease, myocardial infarction, and pneumonia are gathered.

TL;DR: The relation of GLN to reduced glutathione metabolism is described and the alteration of reduced glutATHione metabolism under a variety of clinical conditions such as reperfusion injury, myocardial infarction, respiratory insufficiency, cancer, diabetes, liver disease, and clinical protein catabolism are discussed.

TL;DR: The current knowledge of putative biomarkers is presented, focusing on their application to the major physiological mechanisms affected by the aging process including physical capability, nutritional status, body composition, endocrine and immune function.

TL;DR: A narrative review summarizes the current body of knowledge on (i) the composition and structure of the ECM, (ii) molecular pathways involved in ECM remodeling, (iii) the physiological roles of muscle ECM), (iv) dysregulations of ECM with aging and disease as well as (v) the adaptations of Muscle ECM to training and disuse.