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Benson T. Akingbemi
Researcher at Auburn University
Publications - 45
Citations - 4546
Benson T. Akingbemi is an academic researcher from Auburn University. The author has contributed to research in topics: Testosterone & Leydig cell. The author has an hindex of 24, co-authored 42 publications receiving 4210 citations. Previous affiliations of Benson T. Akingbemi include Population Council & University of Alabama.
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Journal ArticleDOI
In vitro molecular mechanisms of bisphenol A action.
Yelena B. Wetherill,Benson T. Akingbemi,Jun Kanno,John A. McLachlan,Angel Nadal,Carlos Sonnenschein,Cheryl S. Watson,R. Thomas Zoeller,Scott M. Belcher +8 more
TL;DR: This review is a detailed review of published studies that have focused on the mechanistic basis of BPA action in diverse experimental models and an assessment of the strength of the evidence regarding the published BPA research.
Journal ArticleDOI
Chapel Hill bisphenol A expert panel consensus statement: integration of mechanisms, effects in animals and potential to impact human health at current levels of exposure.
Frederick S. vom Saal,Benson T. Akingbemi,Scott M. Belcher,Linda S. Birnbaum,D. Andrew Crain,Marcus Eriksen,Francesca Farabollini,Louis J. Guillette,Russ Hauser,Jerrold J. Heindel,Shuk-Mei Ho,Patricia A. Hunt,Taisen Iguchi,Susan Jobling,Jun Kanno,Ruth A. Keri,Karen E. Knudsen,Hans Laufer,Gerald A. LeBlanc,Michele Marcus,John A. McLachlan,John Peterson Myers,Angel Nadal,Retha R. Newbold,Nicolás Olea,Gail S. Prins,Catherine A. Richter,Beverly S. Rubin,Carlos Sonnenschein,Ana M. Soto,Chris E. Talsness,John G. Vandenbergh,Laura N. Vandenberg,Debby Walser-Kuntz,Cheryl S. Watson,Wade V. Welshons,Yelena B. Wetherill,R. Thomas Zoeller +37 more
TL;DR: This document is a summary statement of the outcome from he meeting: “Bisphenol A: An Examination of the Relevance of cological, In vitro and Laboratory Animal Studies for Assessng Risks to Human Health” sponsored by both the NIEHS and IDCR at NIH/DHHS.
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Inhibition of Testicular Steroidogenesis by the Xenoestrogen Bisphenol A Is Associated with Reduced Pituitary Luteinizing Hormone Secretion and Decreased Steroidogenic Enzyme Gene Expression in Rat Leydig Cells
TL;DR: Exposure of humans to bisphenol A (BPA), a monomer in polycarbonate plastics and a constituent of resins used in food packaging and dentistry, is significant and further studies are warranted to assess the effects of BPA on male fertility.
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Phthalate-induced Leydig cell hyperplasia is associated with multiple endocrine disturbances.
TL;DR: Investigation of the effects of chronic exposures to low environmentally relevant DEHP levels on testicular function shows that prolonged exposures to this agent induced high levels of the gonadotropin luteinizing hormone and increased the serum concentrations of sex hormones, suggesting the possibility of multiple crosstalks between androgen, estrogen, and steroid hormone receptors.
Journal ArticleDOI
Modulation of Rat Leydig Cell Steroidogenic Function by Di(2-Ethylhexyl)Phthalate
Benson T. Akingbemi,Robert T. Youker,Chantal M. Sottas,Ren-Shan Ge,Emily Katz,Gary R. Klinefelter,Barry R. Zirkin,Matthew P. Hardy +7 more
TL;DR: Data from this study show that DEHP effects on Leydig cell steroidogenesis are influenced by the stage of development at exposure and may occur through modulation of T-biosynthetic enzyme activity and serum LH levels.