B
Bing Zhang
Researcher at Nanjing University
Publications - 375
Citations - 15282
Bing Zhang is an academic researcher from Nanjing University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 58, co-authored 320 publications receiving 11185 citations. Previous affiliations of Bing Zhang include Zhejiang University & University of Wisconsin-Madison.
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circVAMP3 Drives CAPRIN1 Phase Separation and Inhibits Hepatocellular Carcinoma by Suppressing c‐Myc Translation
TL;DR: CircVAMP3 is identified as a significantly downregulated circRNA in HCC tissues and is a potential prognostic indicator for HCC and may serve as a therapeutic target for H CC treatment.
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Transcriptome Profiling Following Neuronal and Glial Expression of ALS-Linked SOD1 in Drosophila
TL;DR: Cell-specific expression of both dSOD1 and G85R was found to influence lifespan, affect sensitivity to hydrogen peroxide, and alter lipid peroxidation levels, which may help elucidate the individual and combined contributions of motoneurons and glia in ALS.
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Mycobacterial dynamin-like protein IniA mediates membrane fission.
Manfu Wang,Manfu Wang,Xiangyang Guo,Xiuna Yang,Xiuna Yang,Bing Zhang,Bing Zhang,Jie Ren,Aijun Liu,Yajun Ran,Yajun Ran,Bing Yan,Fang Chen,Luke W. Guddat,Junjie Hu,Junjie Hu,Jun Li,Jun Li,Zihe Rao +18 more
TL;DR: The authors present the apo and GTP bound crystal structures of Mycobacterium smegmatis IniA, which folds as a bacterial dynamin-like protein and shows that IniB mediates membrane fission in vitro, and suggest that iniA mediates TB drug-resistance through fission activity to maintain plasma membrane integrity.
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Loss of Drosophila melanogaster p21-activated kinase 3 Suppresses Defects in Synapse Structure and Function Caused by spastin Mutations
TL;DR: It is shown that pak3 mutants have only mild synaptic defects at the larval neuromuscular junction, but exhibit a potent genetic interaction with spastin mutations, suggesting that it functions in vivo to promote filopodia during presynaptic terminal arborization.
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Activation of D1R/PKA/mTOR signaling cascade in medial prefrontal cortex underlying the antidepressant effects of l-SPD.
Bing Zhang,Bing Zhang,Fei Guo,Yuqin Ma,Yingcai Song,Rong Lin,Fuyi Shen,Guo-Zhang Jin,Yang Li,Zhiqiang Liu +9 more
TL;DR: A critical role of D1R/PKA/mTOR signaling cascade in the mPFC during the l-SPD mediated antidepressant process is suggested, which may also provide new insights into the role of mesocortical dopaminergic system in antidepressant effects.