Bo Y. Park

TL;DR: Eviologic investigations focused on nongenetic factors have established advanced parental age and preterm birth as ASD risk factors, indicated that prenatal exposure to air pollution and short interpregnancy interval are potentialrisk factors, and suggested the need for further exploration of certain prenatal nutrients, metabolic conditions, and exposure to endocrine-disrupting chemicals.

TL;DR: There is a pronounced increase in the risk of stillbirth with increasing BMI; the association is strongest at early- and late-term gestation periods.

TL;DR: Findings of a positive association in infants whose older ASD-affected siblings were female suggests an androgen-related mechanism that may be dependent on, or related to, genetic liability factors present more often in families containing female ASD cases, however, this initial finding should be interpreted with considerable caution.

TL;DR: Comparison of placental shape features in younger siblings of children with ASD, who themselves are at high ASD risk, to a sample of low risk peers indicates restricted ability to compensate for intrauterine changes.

TL;DR: Excess weight gain was associated with higher risk of stillbirth among obese and morbidly obese women but was protective against stillbirth in lower weight women, and weight demonstrated a protective effect.