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Bryan W. Jones
Researcher at University of Utah
Publications - 111
Citations - 6111
Bryan W. Jones is an academic researcher from University of Utah. The author has contributed to research in topics: Retina & Retinal. The author has an hindex of 32, co-authored 108 publications receiving 5233 citations. Previous affiliations of Bryan W. Jones include Vision-Sciences, Inc. & Moran Eye Center.
Papers
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Journal ArticleDOI
Neural remodeling in retinal degeneration.
TL;DR: Retinal remodeling is not plasticity, but represents the invocation of mechanisms resembling developmental and CNS plasticities and together, neuronal remodeling and the formation of the glial seal may abrogate many cellular and bionic rescue strategies.
Journal ArticleDOI
Familial advanced sleep-phase syndrome: A short-period circadian rhythm variant in humans
Christopher R. Jones,Scott S. Campbell,Stephanie E. Zone,Fred Cooper,Alison Desano,Patricia J. Murphy,Bryan W. Jones,Laura A. Czajkowski,Louis J. Ptáček +8 more
TL;DR: Three kindreds with a profound phase advance of the sleep–wake, melatonin and temperature rhythms associated with a very short τ represent a well-characterized familial circadian rhythm variant in humans and provide a unique opportunity for genetic analysis of human circadian physiology.
Journal ArticleDOI
Retinal remodeling triggered by photoreceptor degenerations.
Bryan W. Jones,Carl B. Watt,Jeanne M. Frederick,Wolfgang Baehr,Ching-Kang Chen,Edward M. Levine,Ann H. Milam,Matthew M. LaVail,Robert E. Marc +8 more
TL;DR: Remodeling in human and rodent retinas is independent of the initial molecular targets of retinal degenerations, including defects in the retinal pigmented epithelium, rhodopsin, or downstream phototransduction elements, and suggests that the neural retina may be more plastic than previously believed.
Journal ArticleDOI
Retinal remodeling during retinal degeneration
Bryan W. Jones,Robert E. Marc +1 more
TL;DR: This formal deafferentation of the neural retina eliminates the intrinsic glutamatergic drive of the sensory retina and, perhaps more importantly, removes coordinated Ca++-coupled signaling to the Neural retina.
Journal ArticleDOI
Neural reprogramming in retinal degeneration.
Robert E. Marc,Bryan W. Jones,James R. Anderson,Krista Kinard,David W. Marshak,John H. Wilson,Theodore G. Wensel,Robert J. Lucas +7 more
TL;DR: An instance of human RP provides evidence that rod bipolar cell dendrite switching likely triggers new gene expression patterns and may impair cone pathway function, and Focal cone-sparing can preserve iGluR display by nearby bipolar cells, which may facilitate late RP photoreceptor transplantation attempts.