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Byoung-Chan Kim

Researcher at Korea Research Institute of Bioscience and Biotechnology

Publications -  49
Citations -  1211

Byoung-Chan Kim is an academic researcher from Korea Research Institute of Bioscience and Biotechnology. The author has contributed to research in topics: Genome & Whole genome sequencing. The author has an hindex of 16, co-authored 49 publications receiving 1004 citations. Previous affiliations of Byoung-Chan Kim include Yonsei University & Korea University of Science and Technology.

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Native-feather degradation by Fervidobacterium islandicum AW-1, a newly isolated keratinase-producing thermophilic anaerobe

TL;DR: The enzyme from F. islandicum AW-1 is a novel, thermostable keratinolytic serine protease that showed higher specific activity for the keratinous substrates than other proteases and catalyzed the cleavage of peptide bonds more rapidly following the reduction of disulfide bridges in feather keratin by 10 mM dithiothreitol.
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Cloning, expression and characterization of l-arabinose isomerase from Thermotoga neapolitana: bioconversion of d-galactose to d-tagatose using the enzyme

TL;DR: Gene araA encoding an L-arabinose isomerase (AraA) from the hyperthermophile, Thermotoga neapolitana 5068 was cloned, sequenced, and expressed in Escherichia coli, where a 68% conversion of D-galactose to D-tagatose was obtained using the recombinant enzyme at the isomerization temperature of 80 degrees C.
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Purification and characterization of two distinct thermostable lipases from the gram-positive thermophilic bacterium Bacillus thermoleovorans ID-1

TL;DR: Two distinct thermostable lipases were purified to homogeneity and their maximal activities were found with tricaprylin (C8) as a substrate, although both of the enzymes showed different substrate specificities.
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Elucidation of Akkermansia muciniphila Probiotic Traits Driven by Mucin Depletion.

TL;DR: Min content in the growth medium plays a critical role in the improvement by A. muciniphila of high-fat diet-induced obesity, intestinal inflammation, and compromised intestinal barrier integrity related to a decrease in goblet cell density.
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Dysbiosis-induced IL-33 contributes to impaired antiviral immunity in the genital mucosa

TL;DR: It is demonstrated that dysbiosis within the vaginal microbiota results in severe impairment of antiviral protection against herpes simplex virus type 2 infection, and the findings suggest a previously unstudied role of commensal bacteria in the effector phase of the antiviral immune response against genital herpes.