C
Carlo Cervellati
Researcher at University of Ferrara
Publications - 133
Citations - 3155
Carlo Cervellati is an academic researcher from University of Ferrara. The author has contributed to research in topics: Medicine & Oxidative stress. The author has an hindex of 24, co-authored 112 publications receiving 2369 citations.
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Journal ArticleDOI
Decreased arylesterase activity of paraoxonase-1 (PON-1) might be a common denominator of neuroinflammatory and neurodegenerative diseases
Massimiliano Castellazzi,Alessandro Trentini,Arianna Romani,Giuseppe Valacchi,Tiziana Bellini,Gloria Bonaccorsi,Enrico Fainardi,Carlotta Cavicchio,Angelina Passaro,Giovanni Zuliani,Carlo Cervellati +10 more
TL;DR: The data suggest that a depressed arylesterase activity could be a common denominator of different neurological diseases which, independently of their peculiar ethiopathogenesis and pathophysiology, appear to be all characterized by an altered systemic redox balance.
Journal ArticleDOI
Serum paraoxonase and arylesterase activities of paraoxonase-1 (PON-1), mild cognitive impairment, and 2-year conversion to dementia: A pilot study.
Carlo Cervellati,Alessandro Trentini,Arianna Romani,Tiziana Bellini,Cristina Bosi,Beatrice Ortolani,Amedeo Zurlo,Angelina Passaro,Davide Seripa,Giovanni Zuliani +9 more
TL;DR: The results suggest that in MCI patients low PON‐1 activity might contribute to identify individuals susceptible to develop vascular dementia.
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Oxidative stress as a possible pathogenic cofactor of post-menopausal osteoporosis: Existing evidence in support of the axis oestrogen deficiency-redox imbalance-bone loss.
TL;DR: A critical overview of the most significant epidemiological studies on the effects of dietary antioxidants on bone health and to devise a strategy to overcome the limitations emerged and controversial results are provided.
Journal ArticleDOI
Keratinocytes oxidative damage mechanisms related to airbone particle matter exposure.
Arianna Romani,Carlo Cervellati,Ximena M. Muresan,Giuseppe Belmonte,Alessandra Pecorelli,Franco Cervellati,Mascia Benedusi,Pablo Evelson,Giuseppe Valacchi,Giuseppe Valacchi +9 more
TL;DR: Overall these data, suggest lipid and protein oxidative damage, as well as an increase of inflammatory response after being challenged with CAPs.
Journal ArticleDOI
Impaired enzymatic defensive activity, mitochondrial dysfunction and proteasome activation are involved in RTT cell oxidative damage.
Carlo Cervellati,Claudia Sticozzi,Arianna Romani,Giuseppe Belmonte,Domenico De Rasmo,Anna Signorile,Franco Cervellati,Chiara Milanese,Pier G. Mastroberardino,Alessandra Pecorelli,Vinno Savelli,Henry Jay Forman,Joussef Hayek,Giuseppe Valacchi,Giuseppe Valacchi +14 more
TL;DR: The findings suggest that the systemic OS levels in RTT can be a consequence of both: increased endogenous oxidants as well as altered mitochondrial biogenesis with a decreased activity of defensive enzymes that leads to posttranslational oxidant protein modification and a proteasome activity impairment.