C
Christina Wang
Researcher at Los Angeles Biomedical Research Institute
Publications - 450
Citations - 30621
Christina Wang is an academic researcher from Los Angeles Biomedical Research Institute. The author has contributed to research in topics: Testosterone (patch) & Sperm. The author has an hindex of 88, co-authored 422 publications receiving 27941 citations. Previous affiliations of Christina Wang include Queen Mary University of London & Cedars-Sinai Medical Center.
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Journal ArticleDOI
Minocycline up-regulates BCL-2 levels in mitochondria and attenuates male germ cell apoptosis
Mark Castanares,Yanira Vera,Krista Erkkilä,Sauli Kyttänen,Yanhe Lue,Leo Dunkel,Christina Wang,Ronald S. Swerdloff,Amiya P. Sinha Hikim +8 more
TL;DR: Minocycline-mediated protection occurred at the mitochondria, involving the restoration of the BCL-2 levels and, in turn, suppression of cytochrome c and DIABLO release.
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Azoospermia: virtual reality or possible to quantify?
Trevor G. Cooper,B. Hellenkemper,J. Jonckheere,Nico Callewaert,Arijan Grootenhuis,W. M. Kersemaekers,Andrew Leung,Christina Wang +7 more
TL;DR: The accurate measurement of low sperm counts is facilitated by the use of large-volume chambers and fluorescence microscopy, and this permits the definition of lower limits of sperm concentrations for azoospermic samples.
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Proteomic analysis of testis biopsies in men treated with transient scrotal hyperthermia reveals the potential targets for contraceptive development.
Hui Zhu,Yugui Cui,Jin Xie,Ling Chen,Xiangxiang Chen,Xuejiang Guo,Yefei Zhu,Xing-Hai Wang,Jian-Sun Tong,Zuomin Zhou,Yue Jia,Yanhe Lue,Amiya P. Sinha Hikim,Christina Wang,Ronald S. Swerdloff,Jiahao Sha +15 more
TL;DR: It was found that HNRNPH1 was an anti‐apoptosis protein that could regulate the expression of other heat‐induced proteins that were found to be key molecular targets affecting spermatogenesis after heat treatment.
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Exposure to prenatal life events stress is associated with masculinized play behavior in girls
TL;DR: Findings in humans and animal models suggesting that prenatal stress is a non-chemical endocrine disruptor that may have androgenic effects on female fetuses and anti-androgenic effect on male fetuses are confirmed.
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Pituitary-thyroid responses to surgical stress.
TL;DR: The effect of surgery on pituitary-thyroid function was studied in 12 euthyroid patients, with a sharp early increase in total thyroxine level, causing displacement of triiodothyronine from thyroid hormone binding proteins resulting in the elevation of the biologically more potent free triiodoryronine fraction.