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David Barton

Researcher at Baker IDI Heart and Diabetes Institute

Publications -  60
Citations -  3150

David Barton is an academic researcher from Baker IDI Heart and Diabetes Institute. The author has contributed to research in topics: Major depressive disorder & Panic disorder. The author has an hindex of 28, co-authored 58 publications receiving 2780 citations. Previous affiliations of David Barton include University of Melbourne & Royal Melbourne Hospital.

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Sertraline treatment of major depression in patients with acute MI or unstable angina

TL;DR: It is suggested that sertraline is a safe and effective treatment for recurrent depression in patients with recent MI or unstable angina and without other life-threatening medical conditions.
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Depression and the Link with Cardiovascular Disease

TL;DR: This review provides an outline of the association between major depressive disorder (MDD) and coronary heart disease (CHD), and the activation of stress pathways have been implicated as a neurochemical mechanism that links MDD and CHD.
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Sympathetic activity in major depressive disorder: identifying those at increased cardiac risk?

TL;DR: A subset of patients with major depressive disorder in whom sympathetic nervous activity is extraordinarily high, including in the sympathetic outflow to the heart is identified, which may reduce sympathetic activity in a manner likely to reduce cardiac risk.
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White matter hyperintensities in vascular contributions to cognitive impairment and dementia (VCID): Knowledge gaps and opportunities

Jessica Alber, +53 more
TL;DR: Outstanding questions about white matter hyperintensities and their relation to cognition, dementia, and AD are identified and answered to improve prevention and treatment of WMHs and dementia.
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Elevated brain serotonin turnover in patients with depression: effect of genotype and therapy.

TL;DR: In this article, the effect of the 5-HTT genotype on brain serotonin turnover was evaluated and the influence of SSRI therapy on serotonin turnover is investigated, which suggests that high brain serotonin may be a biological substrate of major depressive disorder.