D
David E. Weinstein
Researcher at Albert Einstein College of Medicine
Publications - 21
Citations - 745
David E. Weinstein is an academic researcher from Albert Einstein College of Medicine. The author has contributed to research in topics: Regeneration (biology) & Schwann cell. The author has an hindex of 9, co-authored 21 publications receiving 711 citations.
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Journal ArticleDOI
Fas engagement induces neurite growth through ERK activation and p35 upregulation.
Julie Desbarats,Julie Desbarats,Julie Desbarats,Raymond B. Birge,Raymond B. Birge,Manuelle Mimouni-Rongy,David E. Weinstein,Jean-Sébastien Palerme,M. Karen Newell +8 more
TL;DR: It is reported that crosslinking Fas on primary sensory neurons induces neurite growth through sustained activation of the extracellular-signal regulated kinase (ERK) pathway and the consequent upregulation of p35, a mediator of neurite outgrowth.
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Caveolin-1 Expression Inhibits Wnt/β-Catenin/Lef-1 Signaling by Recruiting β-Catenin to Caveolae Membrane Domains
Ferruccio Galbiati,Daniela Volonté,Anthony M. C. Brown,Anthony M. C. Brown,David E. Weinstein,Avri Ben-Ze'ev,Richard G. Pestell,Michael P. Lisanti +7 more
TL;DR: It is suggested that caveolin-1 expression can modulate Wnt/β-catenin/Lef-1 signaling by regulating the intracellular localization of β-Catenin.
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CD81 Regulates Neuron-Induced Astrocyte Cell-Cycle Exit
TL;DR: It is shown that CD81 is expressed on the surface of the astrocyte and that its expression level can be modulated by contact with neurons, and that there is a unique domain, recognized by Eat1, that is required for astroCyte cell-cycle withdrawal in response to neurons.
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The POU Gene Brn-5 Is Induced by Neuregulin and Is Restricted to Myelinating Schwann Cells
TL;DR: The developmental expression patterns of Brn-5 and SCIP are inverse, with Brn -5 stably expressed in the adult myelinating Schwann cell, but virtually absent during promyelination, and it is shown that the induction of the two genes is independent.
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Isolation and Purification of Primary Rodent Astrocytes
TL;DR: The first isolation procedure described in this unit takes advantage of the proliferative ability of these cells, as does the second, except that no antibody or complement treatment is required.