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Dawei Guo

Researcher at Nanjing Agricultural University

Publications -  46
Citations -  944

Dawei Guo is an academic researcher from Nanjing Agricultural University. The author has contributed to research in topics: Medicine & Maduramicin. The author has an hindex of 13, co-authored 36 publications receiving 705 citations. Previous affiliations of Dawei Guo include Southeast University.

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Anti-leukemia activity of PVP-coated silver nanoparticles via generation of reactive oxygen species and release of silver ions

TL;DR: It is found that AgNPs could inhibit the viability of AML cells including the isolates from AML patients, and supported the model that both generation of ROS and release of silver ions played critical roles in the AgnPs-induced cytotoxic effect against AMl cells.
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Controllable synthesis and biomedical applications of silver nanomaterials.

TL;DR: The controllable synthesis of silver nanomaterials including nanorods, nanowires, nanotubes, nanoprisms, nanoplates, nanodisks, nanospheres, and nanopolyhedrons, etc are reviewed.
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Shape-controlled fabrication of magnetite silver hybrid nanoparticles with high performance magnetic hyperthermia

TL;DR: It is demonstrated that the hybridisation of Fe3O4 and Ag NPs could greatly enhance the magnetic hyperthermia efficiency of Fe2O4 NPs, and can be used to be as high performance magnetichyperthermia mediators based on a simple and effective preparation approach.
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A caffeic acid mediated facile synthesis of silver nanoparticles with powerful anti-cancer activity.

TL;DR: Results showed that the AgNPs could rapidly and simply be synthesized using caffeic acid as both a reducing agent and stabilizer, as well as being stable in aqueous solution and could enter cells and effectively inhibit viability of tumor cells via induction of apoptosis.
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Activation of autophagy by elevated reactive oxygen species rather than released silver ions promotes cytotoxicity of polyvinylpyrrolidone-coated silver nanoparticles in hematopoietic cells

TL;DR: It is suggested that autophagy is involved in the cytotoxicity of PVP-coated AgNPs in normal hematopoietic cells, and the inhibition of autophagic flux is a novel and potent strategy to protect normal heMatopoetic cells upon treatment with AgNNP.