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Elizabeth J. Atkinson

Researcher at Mayo Clinic

Publications -  339
Citations -  32583

Elizabeth J. Atkinson is an academic researcher from Mayo Clinic. The author has contributed to research in topics: Population & Osteoporosis. The author has an hindex of 88, co-authored 321 publications receiving 29080 citations. Previous affiliations of Elizabeth J. Atkinson include University of British Columbia & University of Rochester.

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Incidence of clinically diagnosed vertebral fractures: a population-based study in Rochester, Minnesota, 1985-1989.

Colin Cooper, +3 more
- 03 Dec 2009 - 
TL;DR: Investigation of vertebral fractures in Rochester, Minnesota found that fractures following moderate trauma were higher in women than in men and rose steeply with age in both genders, while fractures following severe trauma were more frequent in men, and their incidence increased less with age.
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Population-Based Study of Survival after Osteoporotic Fractures

Cyrus Cooper, +4 more
- 01 May 1993 - 
TL;DR: The survival rate of 335 residents of Rochester, Minnesota, who had an initial radiologic diagnosis of vertebral fracture between 1985 and 1989 was worse than expected, and diverged steadily from expected values throughout the course of the study.

An Introduction to Recursive Partitioning Using the RPART Routines

Terry M. Therneau, +1 more
TL;DR: The tree is constructed: Splitting criteria, building the tree, variable importance, and more.
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Relationship of serum sex steroid levels and bone turnover markers with bone mineral density in men and women: A key role for bioavailable estrogen

Sundeep Khosla, +5 more
- 01 Jul 1998 - 
TL;DR: It is shown that age-related bone loss may be the result of E deficiency not just in postmenopausal women, but also in men, and bioavailable E levels decline significantly with age and are important predictors of BMD in men as well as women.
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Cellular senescence mediates fibrotic pulmonary disease

Marissa J. Schafer, +22 more
- 23 Feb 2017 - 
TL;DR: It is demonstrated that early-intervention suicide-gene-mediated senescent cell ablation improves pulmonary function and physical health, although lung fibrosis is visibly unaltered, and fibrotic lung disease is mediated, in part, by senescent cells.