E
Ellen S. Morgan
Researcher at Beth Israel Deaconess Medical Center
Publications - 56
Citations - 3495
Ellen S. Morgan is an academic researcher from Beth Israel Deaconess Medical Center. The author has contributed to research in topics: Mast cell & Degranulation. The author has an hindex of 29, co-authored 56 publications receiving 3359 citations. Previous affiliations of Ellen S. Morgan include Technion – Israel Institute of Technology & Harvard University.
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Journal ArticleDOI
Basophils Produce IL-4 and Accumulate in Tissues after Infection with a Th2-inducing Parasite
Booki Min,Melanie Prout,Jane Hu-Li,Jinfang Zhu,Dragana Jankovic,Ellen S. Morgan,Joseph F. Urban,Ann M. Dvorak,Fred D. Finkelman,Graham LeGros,William E. Paul +10 more
TL;DR: Infection with a parasite that induces a “Th2-type response” resulted in accumulation of tissue basophils, and these cells, stimulated by a non-FcR cross-linking mechanism, are a principal source of in vivo IL-4 production.
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Heterogeneity of the Angiogenic Response Induced in Different Normal Adult Tissues by Vascular Permeability Factor/Vascular Endothelial Growth Factor
Anna Pettersson,Janice A. Nagy,Lawrence F. Brown,Christian Sundberg,Ellen S. Morgan,Jungles S,Carter R,José Eduardo Krieger,José Eduardo Krieger,Eleanor J. Manseau,V. S. Harvey,I A Eckelhoefer,Dian Feng,Ann M. Dvorak,Richard C. Mulligan,Harold F. Dvorak +15 more
TL;DR: The muscular vessels that developed from mother vessels in skin and perimuscle fat have the structure of collaterals and could be useful clinically in the relief of tissue ischemia and indicate that the angiogenic response induced by VPF/VEGF is heterogeneous and tissue specific.
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The vesiculo-vacuolar organelle (VVO): a distinct endothelial cell structure that provides a transcellular pathway for macromolecular extravasation.
TL;DR: The findings indicate that VVOs provide a major pathway for the extravasation of circulating macromolecules across endothelia taller than capillary endothelium and suggest that upregulated VVO function accounts for the well‐known hyperpermeability of tumor blood vessels.
Journal Article
Morphology of delayed type hypersensitivity reactions in man. I. Quantitative description of the inflammatory response.
Harold F. Dvorak,Martin C. Mihm,Ann M. Dvorak,Richard A. Johnson,Eleanor J. Manseau,Ellen S. Morgan,Robert B. Colvin +6 more
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Eosinophil Lipid Bodies: Specific, Inducible Intracellular Sites for Enhanced Eicosanoid Formation
TL;DR: The specific intracellular sites at which enzymes act to generate arachidonate-derived eicosanoid mediators of inflammation are uncertain this paper, however, it is known that platelet-activating factor (PAF)-induced increases in lipid body numbers correlated with enhanced production of both lipoxygenase-and cyclooxygenases-derived EICosanoids.