E
Erica di Martino
Researcher at University of Leeds
Publications - 15
Citations - 613
Erica di Martino is an academic researcher from University of Leeds. The author has contributed to research in topics: Bladder cancer & Referral. The author has an hindex of 7, co-authored 12 publications receiving 526 citations. Previous affiliations of Erica di Martino include St James's University Hospital.
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Journal ArticleDOI
Distinct MicroRNA Alterations Characterize High- and Low-Grade Bladder Cancer
James W.F. Catto,Saiful Miah,Helen C. Owen,Helen E. Bryant,Katie N. Myers,Ewa Dudziec,Stéphane Larré,Marta Milo,Ishtiaq Rehman,Derek J. Rosario,Erica di Martino,Margaret A. Knowles,Mark Meuth,Adrian L. Harris,Freddie C. Hamdy +14 more
TL;DR: It is found that altered microRNA expression is common in U CC and occurs early in tumorogenesis, and distinct microRNA alterations characterize UCC and target genes in a pathway-specific manner.
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A Decade of FGF Receptor Research in Bladder Cancer: Past, Present, and Future Challenges
TL;DR: Overall, FGFRs hold promise as therapeutic targets, diagnostic and prognostic markers, and screening tools for early detection and clinical management of UC.
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The NAD(P)H:quinone oxidoreductase I C609T polymorphism modifies the risk of Barrett esophagus and esophageal adenocarcinoma.
Erica di Martino,Laura J. Hardie,Christopher P. Wild,Yun Y Gong,J. R. Olliver,Martin D Gough,Nigel C. Bird +6 more
TL;DR: Overall, the results of this study suggest that the NQO1 TT genotype may offer protection from reflux complications such as Barrett esophagus and esophageal adenocarcinoma.
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Matrix metalloproteinases in keratoconus – Too much of a good thing?
TL;DR: The current knowledge on the role of MMPs in the pathogenesis of keratoconus is summarized, with extensive evidence that M MPs are upregulated in the corneal tissue and tears of KC patients, implicating dysregulated proteolysis in KC, with an increase in the level of some MMPS, particularly MMP‐1 and MMP-9, confirmed in multiple independent studies.
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FGFR3 mutation affects cell growth, apoptosis and attachment in keratinocytes.
Christian Hafner,Erica di Martino,Eva Pitt,Thomas Stempfl,Darren C. Tomlinson,Arndt Hartmann,Michael Landthaler,Margaret A. Knowles,Thomas Vogt +8 more
TL;DR: The results suggest that an increased cell number at confluence along with a decreased apoptosis may contribute to the development of acanthotic tumors in FGFR3 mutant skin in vivo.