E
Erwin G. Van Meir
Researcher at University of Alabama at Birmingham
Publications - 202
Citations - 39049
Erwin G. Van Meir is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Glioma & Cancer. The author has an hindex of 75, co-authored 183 publications receiving 32866 citations. Previous affiliations of Erwin G. Van Meir include Emory University & University Hospital of Lausanne.
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Journal ArticleDOI
Opening the chamber of peer-review secrets
TL;DR: In the spirit of science, the authors should ask why studies don't reflect farmers' experiences.
Patent
Inhibitors of hif and angiogenesis
Erwin G. Van Meir,Binghe Wang +1 more
TL;DR: Inhibitors of the Hypoxia Inducible Factor (HIF) and angiogenesis and their methods of use including cancer, hypoxia related pathologies, disorders leading to ischemia, for example stroke and ischemic heart disease, and non-cancerous angiogenic diseases are provided.
Journal ArticleDOI
p14ARF suppresses tumor-induced thrombosis by regulating the tissue factor pathway
TL;DR: The results identify the critical signaling pathways activated by P14ARF to prevent vascular microthrombosis triggered by glioma cells and Stimulation of this pathway might be used as a therapeutic strategy to reduce aggressive phenotypes associated with necrotic tumors, including glioblastoma.
Journal ArticleDOI
A conspiracy of glioma and endothelial cells to invade the normal brain.
TL;DR: This data indicates that pre-operatively BRAF checkpoint blockade-induction-guided cell reprograming is a feasible and effective treatment for central giant cell granuloma.
Journal ArticleDOI
Targeting HIF-activated collagen prolyl 4-hydroxylase expression disrupts collagen deposition and blocks primary and metastatic uveal melanoma growth
Stefan Kaluz,Qing Zhang,Qing Zhang,Yuki Kuranaga,Hua Yang,Satoru Osuka,Satoru Osuka,Debanjan Bhattacharya,Narra S. Devi,Jiyoung Mun,Wei Wang,Ruiwen Zhang,Mark M. Goodman,Hans E. Grossniklaus,Erwin G. Van Meir +14 more
TL;DR: It is demonstrated that extracellular collagen matrix formation can be targeted in UM by inhibiting hypoxia-induced P4HA1 and P 4HA2 expression, warranting further development of this strategy in patients with uveal melanoma.