E
Erwin G. Van Meir
Researcher at University of Alabama at Birmingham
Publications - 202
Citations - 39049
Erwin G. Van Meir is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Glioma & Cancer. The author has an hindex of 75, co-authored 183 publications receiving 32866 citations. Previous affiliations of Erwin G. Van Meir include Emory University & University Hospital of Lausanne.
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Journal ArticleDOI
Are Risk Factors for Growth of Choroidal Nevi Associated With Malignant Transformation? Assessment With a Validated Genomic Biomarker.
Posted ContentDOI
The epigenetic evolution of gliomas is determined by their IDH1 mutation status and treatment regimen
Tathiane M. Malta,Thais S. Sabedot,Indrani Datta,Luciano Garofano,Wies Vallentgoed,Frederick S. Varn,Kenneth Aldape,Fulvio D'Angelo,Spyridon Bakas,Jill S. Barnholtz-Sloan,Hui K Gan,Mohammad Hasanain,Ann-Christin Hau,Kevin C. Johnson,Mustafa Khasraw,Emre Kocakavuk,Mathilde C.M. Kouwenhoven,Simona Migliozzi,Simone P. Niclou,Johanna M. Niers,D. Ryan Ormond,Sun Ha Paek,Guido Reifenberger,Peter A. E. Sillevis Smitt,Marion Smits,Lucy F. Stead,Martin J. van den Bent,Erwin G. Van Meir,Annemiek M E Walenkamp,Tobias Weiss,Michael Weller,Bart A. Westerman,Bauke Ylstra,Pieter Wesseling,Anna Lasorella,Pim J. French,Laila M. Poisson,Roel G.W. Verhaak,Antonio Iavarone,Houtan Noushmehr +39 more
TL;DR: In this paper, the authors performed an epigenomic analysis of 143 matched initial and recurrent patients with IDH-wildtype (IDHwt) and IDHmutant gliomas.
Patent
Glioblastoma cell lines that produce angiogenesis inhibiting factor
TL;DR: In this article, a glioblastoma derived angiogenesis inhibiting factor is described, induced in the presence of wild type p53, but not by several mutated forms of p53.
Journal ArticleDOI
Cs-38bai1 is a new tumor suppressor in medulloblastoma.
TL;DR: It is demonstrated that small molecule epigenetic inhibitors of either EZH2 or MBD2 can reactivate BAI1 expression and this provides insight into the neurobiological mechanisms underlying MB tumorigenesis and provides the rationale for the use of epigenetic therapeutics against MB.
Journal ArticleDOI
Generation and initial characterization of mice lacking full length BAI3 (ADGRB3) expression.
Fu Hung Shiu,Jennifer C. Wong,Debanjan Bhattacharya,Yuki Kuranaga,Rashed R Parag,Haifa Alsharif,Sushant Bhatnagar,Erwin G. Van Meir,Andrew Escayg +8 more
TL;DR: In this paper , the authors used CRISPR/Cas9 editing to generate a mouse line with a 7 base pair deletion in Adgrb3 exon 10, which demonstrated reduced brain and body weights and deficits in social interaction.