F
Feng Xue
Researcher at Shanghai Jiao Tong University
Publications - 46
Citations - 1889
Feng Xue is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Liver transplantation & Liver function. The author has an hindex of 17, co-authored 42 publications receiving 1586 citations.
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Journal ArticleDOI
Correction: Corrigendum: Differential developmental requirement and peripheral regulation for dermal Vγ4 and Vγ6T17 cells in health and inflammation
Yihua Cai,Feng Xue,Christopher M. Fleming,Jie Yang,Chuanlin Ding,Yunfeng Ma,Min Liu,Huang-Ge Zhang,Jie Zheng,Na Xiong,Jun Yan +10 more
TL;DR: Huang-Ge Zhang as discussed by the authors was supported by a Research Career Scientist (RCS) Award, but the financial support for this article was not fully acknowledged by the RCS.
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Cisplatin-induced downregulation of miR-199a-5p increases drug resistance by activating autophagy in HCC cell
TL;DR: It is found that miR-199a-5p levels were significantly reduced in HCC patients treated with cisplatin-based chemotherapy, thus offering a new target for chemotherapy of HCC.
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Characteristics, Diagnosis and Prognosis of Acute-on-Chronic Liver Failure in Cirrhosis Associated to Hepatitis B.
Hai Li,L. Chen,Nan-nan Zhang,Shuting Li,Bo Zeng,Marco Pavesi,Alex Amoros,Rajeshwar P. Mookerjee,Qian Xia,Feng Xue,Xiong Ma,Jing Hua,Li Sheng,De-kai Qiu,Qing Xie,Graham R. Foster,Geoffrey Dusheiko,Richard Moreau,Pere Ginès,Vicente Arroyo,Rajiv Jalan +20 more
TL;DR: Current study indicates that ACLF is a clinically and pathophysiology distinct even in HBV patients, and diagnostic criteria, prognostic scores and probably the management of ACLF should base on similar principles.
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Upregulated miR-130a increases drug resistance by regulating RUNX3 and Wnt signaling in cisplatin-treated HCC cell.
TL;DR: It is demonstrated that upregulated miR-130a directly inhibited expression of tumor suppressor gene RUNX3, which resulted in activation of Wnt/β-catenin signaling and increased drug resistance, thus offering a new target for chemotherapy of HCC.
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Monoamine oxidase A suppresses hepatocellular carcinoma metastasis by inhibiting the adrenergic system and its transactivation of EGFR signaling.
Jun Li,Xiao-Mei Yang,Ya-Hui Wang,Mingxuan Feng,Xiao-Jin Liu,Yan-Li Zhang,Shuo Huang,Zheng Wu,Feng Xue,Wenxin Qin,Jianren Gu,Qiang Xia,Zhigang Zhang +12 more
TL;DR: The results of this study may provide insights into the application of MAOA as a novel predictor of clinical outcomes and indicate that increasing MAOA expression or enzyme activity may be a new approach that can be used for HCC treatment.