F
Francesco E. Pontieri
Researcher at Sapienza University of Rome
Publications - 140
Citations - 8326
Francesco E. Pontieri is an academic researcher from Sapienza University of Rome. The author has contributed to research in topics: Parkinson's disease & Dopaminergic. The author has an hindex of 35, co-authored 128 publications receiving 7542 citations. Previous affiliations of Francesco E. Pontieri include National Institutes of Health & University of Cagliari.
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Journal ArticleDOI
Effects of haloperidol on the expression of lymphocyte dopamine receptor mRNAs in the rat.
Brunella Caronti,Francesca Passarelli,Caterina Calderaro,Guido Palladini,Francesco E. Pontieri +4 more
TL;DR: The results indicate short-lasting dynamic changes of expression of lymphocyte D1B dopamine receptor mRNA by haloperidol and suggest that the effects of dopamine and dopaminergic drugs on the immune system might be mediated, at least in part, by direct interaction of these substances with dopamine receptors on lymphocyte membrane.
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Psychiatric profile of motor subtypes of de novo drug-naïve Parkinson's disease patients
Francesca Assogna,Clelia Pellicano,Luca Cravello,Cinzia Savini,Mariangela Pierantozzi,Bruno Mercuri,Carlo Caltagirone,Francesco E. Pontieri,Gianfranco Spalletta,Alessandro Stefani +9 more
TL;DR: This study highlights the need to understand more fully the rationale behind the continued heterogeneity in the development of different motor subtypes of Parkinson's disease.
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Levodopa/Carbidopa Intestinal Gel for Treatment of Advanced Parkinson’s Disease: An Update on the Effects of Cognitive Functions
TL;DR: The results from several preliminary studies suggest that efficacy of LCGI on motor symptoms may be accompanied by good tolerability and potential benefit on several non-motor symptoms, including cognitive impairment, in advanced PD patients.
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Electroconvulsive shock blocks the opioid-mediated inhibition of dopamine release in rat striatal slices
TL;DR: The results demonstrate that pre-treatment with ECS suppresses the inhibition of dopamine release mediated by κ opioid receptors, and suggest that single ECS treatment modifies the sensitivity of the λ opioid receptors located on the presynatic dopamine terminals in the rat striatum.
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Selective metabolic activation by apomorphine in striosomes of denervated striatum in MPTP-induced hemiparkinsonian monkeys.
TL;DR: Apomorphine treatment was found to result in higher rates of glucose utilization in the denervated striatum than in the intact hemisphere, providing evidence for differential functional and/or metabolic regulation in striatal striosomes and matrix in parkinsonian syndromes.