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Francesco Latrofa

Researcher at University of Pisa

Publications -  82
Citations -  2935

Francesco Latrofa is an academic researcher from University of Pisa. The author has contributed to research in topics: Thyroid & Thyroglobulin. The author has an hindex of 26, co-authored 67 publications receiving 2253 citations. Previous affiliations of Francesco Latrofa include World Health Organization & University of California, Los Angeles.

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Hashimoto's thyroiditis is associated with papillary thyroid carcinoma: role of TSH and of treatment with l-thyroxine

TL;DR: The frequency of PTC is significantly higher in nodular-HT than in NG and is associated with increased levels of serum TSH, and treatment with L-T4 reduces TSH levels and decreases the occurrence of clinically detectable PTC.
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Is Subacute Thyroiditis an Underestimated Manifestation of SARS-CoV-2 Infection? Insights From a Case Series.

TL;DR: Clinical, biochemical and imaging features of the 4 patients with SAT related to SARS-CoV-2 infection are described to alert physicians that SAT may be an underestimated manifestation of COVID-19.
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Outcome of Thyroid Function in Graves’ Patients Treated with Radioiodine: Role of Thyroid-Stimulating and Thyrotropin-Blocking Antibodies and of Radioiodine-Induced Thyroid Damage

TL;DR: The early outcome of thyroid function after 131I for GD is mainly related to pretreatment thyroid volume and to the degree of its reduction after therapy; high TSAb levels before 131I are associated with a relative resistance to therapy; a postradioiodine increase in TSAb Levels is related to the development of hypothyroidism.
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Role of genetic and non-genetic factors in the etiology of Graves’ disease

TL;DR: The respective role of genetic and non-genetic factors in the etiology of Graves’ disease are reviewed, taking advantage of the great bulk of data generated especially over the past 30 years.
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Spontaneous improvement of untreated mild Graves' ophthalmopathy: Rundle's curve revisited

TL;DR: In confirmation of Rundle's observations, untreated GO improves spontaneously with time in the majority of patients, with an activity peak between 13 and 24 months, which may have implications in determining the proper timing of GO treatments.