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Franco De Michieli

Researcher at University of Turin

Publications -  28
Citations -  2627

Franco De Michieli is an academic researcher from University of Turin. The author has contributed to research in topics: Insulin resistance & Nonalcoholic fatty liver disease. The author has an hindex of 20, co-authored 27 publications receiving 2150 citations.

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Dietary habits and their relations to insulin resistance and postprandial lipemia in nonalcoholic steatohepatitis

TL;DR: Dietary habits may promote steatohepatitis directly by modulating hepatic triglyceride accumulation and antioxidant activity as well as indirectly by affecting insulin sensitivity and postprandial triglyceride metabolism.
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Changes in Weight and Nutritional Habits in Adults with Obesity during the "Lockdown" Period Caused by the COVID-19 Virus Emergency.

TL;DR: The adverse mental burden linked to the COVID-19 pandemic was greatly associated with increased weight gain, and individuals with obesity significantly gained weight 1 month after the beginning of the quarantine.
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Adipokines in NASH : Postprandial lipid metabolism as a link between adiponectin and liver disease

TL;DR: Hypoadiponectinemia is present before overt diabetes and obesity appear and correlates with the severity of liver histology in NASH and may have important pathogenetic and therapeutic implications in both liver and metabolic disease.
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Hypoadiponectinemia Predicts the Severity of Hepatic Fibrosis and Pancreatic Beta-Cell Dysfunction in Nondiabetic Nonobese Patients with Nonalcoholic Steatohepatitis

TL;DR: Hypoadiponectinemia is a feature of NASH and may have a pathogenetic role in β-cell dysfunction and in hepatic necroinflammation and fibrosis, independently of insulin resistance, visceral fat accumulation, TNF-α axis activity, and dietary habits.
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Adiponectin gene polymorphisms modulate acute adiponectin response to dietary fat: Possible pathogenetic role in NASH.

TL;DR: The at‐risk adiponectin SNPs 45TT and 276GT are significantly more prevalent in NAFLD than in the general population; they are associated with severity of liver disease, with blunted postprandial adiponECTin response, and with an atherogenic postpr andial lipoprotein profile in NASH independently of fasting adipokine and lipid levels.