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Marilena Durazzo

Researcher at University of Turin

Publications -  143
Citations -  5607

Marilena Durazzo is an academic researcher from University of Turin. The author has contributed to research in topics: Population & Hepatitis. The author has an hindex of 36, co-authored 130 publications receiving 4953 citations. Previous affiliations of Marilena Durazzo include Hochschule Hannover.

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Dietary habits and their relations to insulin resistance and postprandial lipemia in nonalcoholic steatohepatitis

TL;DR: Dietary habits may promote steatohepatitis directly by modulating hepatic triglyceride accumulation and antioxidant activity as well as indirectly by affecting insulin sensitivity and postprandial triglyceride metabolism.
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Different cytokine profiles of intraphepatic T cells in chronic hepatitis B and hepatitis C virus infections

TL;DR: The different cytokine profiles of T cells within the liver in chronic HBV and HCV infections illustrate a different behavior of the local immune response in these two infections that may have pathogenetic implications.
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Adipokines in NASH : Postprandial lipid metabolism as a link between adiponectin and liver disease

TL;DR: Hypoadiponectinemia is present before overt diabetes and obesity appear and correlates with the severity of liver histology in NASH and may have important pathogenetic and therapeutic implications in both liver and metabolic disease.
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Effectiveness of a lifestyle intervention on metabolic syndrome. A randomized controlled trial.

TL;DR: A lifestyle intervention based on general recommendations was effective in reducing multiple metabolic/inflammatory abnormalities and the usual care by family physicians was ineffective at modifying progressive metabolic deterioration in high-risk individuals.
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Hypoadiponectinemia Predicts the Severity of Hepatic Fibrosis and Pancreatic Beta-Cell Dysfunction in Nondiabetic Nonobese Patients with Nonalcoholic Steatohepatitis

TL;DR: Hypoadiponectinemia is a feature of NASH and may have a pathogenetic role in β-cell dysfunction and in hepatic necroinflammation and fibrosis, independently of insulin resistance, visceral fat accumulation, TNF-α axis activity, and dietary habits.