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Franz Fazekas

Researcher at Medical University of Graz

Publications -  634
Citations -  59050

Franz Fazekas is an academic researcher from Medical University of Graz. The author has contributed to research in topics: Hyperintensity & Stroke. The author has an hindex of 101, co-authored 629 publications receiving 49775 citations. Previous affiliations of Franz Fazekas include Hospital of the University of Pennsylvania & University of Graz.

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Journal ArticleDOI

Prevalence of stenoses and occlusions of brain-supplying arteries in young stroke patients

TL;DR: A substantial proportion of atherosclerotic carotid artery stenoses and occlusions in younger stroke patients had symptomatic or asymptomatic large-artery atherosclerosis, which should encourage future stroke prevention campaigns to target risk factor modification in young people.
Book ChapterDOI

Risk factors and progression of small vessel disease-related cerebral abnormalities

TL;DR: The data suggest that drugs influencing the RAS system may allow to intervene with an unfavorable course of cerebral small vessel disease.
Journal ArticleDOI

Vascular risk factors, white matter hyperintensities and hippocampal volume in normal elderly individuals.

TL;DR: The data indicate HV loss in parallel with the whole brain and suggest no specific vulnerability towards vascular risk factors or age-related WMH in a cognitively intact normal elderly population, which supports the utility of HV measurements to identify impending AD.
Journal ArticleDOI

Prevalence of multiple sclerosis in Austria. Results of a nationwide survey.

TL;DR: The prevalence of MS in Austria was found to be similar to that of other countries in Central Europe, and a novel extrapolation model, based on frequencies of patients visits at MS clinics, was used to estimate the overall prevalence.
Journal ArticleDOI

Contribution of Convexal Subarachnoid Hemorrhage to Disease Progression in Cerebral Amyloid Angiopathy

TL;DR: In cerebral amyloid angiopathy, leakage of meningeal vessels seems to be a major cause for recurrent intrasulcal bleedings, which lead to the propagation of cSS and indicate sites with increased vulnerability for future ICH.