F
Franz Fazekas
Researcher at Medical University of Graz
Publications - 634
Citations - 59050
Franz Fazekas is an academic researcher from Medical University of Graz. The author has contributed to research in topics: Hyperintensity & Stroke. The author has an hindex of 101, co-authored 629 publications receiving 49775 citations. Previous affiliations of Franz Fazekas include Hospital of the University of Pennsylvania & University of Graz.
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Angiotensinogen gene promoter haplotype and microangiopathy-related cerebral damage: results of the Austrian Stroke Prevention Study
TL;DR: The B haplotype of the AGT promoter in the absence of the wild-type A haplotype might represent a genetic susceptibility factor for microangiopathy-related cerebral damage.
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Notch1 and its ligand Jagged1 are present in remyelination in a T-cell- and antibody-mediated model of inflammatory demyelination.
TL;DR: It is unlikely, at least in the paradigm of MOG-EAE, that Notch signaling is responsible for a failure of remyelination, as constituents of the Notch pathway are expressed in remyELination in an animal model of T-cell- and antibody-mediated CNS demyelinated.
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White Matter Changes Contribute to Corpus Callosum Atrophy in the Elderly: The LADIS Study
Charlotte Ryberg,Egill Rostrup,Karl Sjöstrand,Olaf B. Paulson,Frederik Barkhof,Philip Scheltens,E.C.W. van Straaten,Franz Fazekas,R. Schmidt,Timo Erkinjuntti,Lars-Olof Wahlund,A. M. Basile,Leonardo Pantoni,Domenico Inzitari,Gunhild Waldemar +14 more
TL;DR: Investigation of the potential correlation between regional age-related white matter changes (ARWMC) and atrophy of corpus callosum in elderly subjects with leukoaraiosis suggested that ARWMC may lead to a gradual loss of CC tissue.
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Diffusion imaging of the human spinal cord and the vertebral column.
Roland Bammer,Franz Fazekas +1 more
TL;DR: The study of diffusion anisotropy may open new avenues for the detection and better understanding of damage to the long fiber tracts with important clinical implications for disorders like multiple sclerosis and amyotrophic lateral sclerosis.
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Lower Levels of N-Acetylaspartate in Multiple Sclerosis Patients With the Apolipoprotein E ϵ4 Allele
Christian Enzinger,Stefan Ropele,S. Strasser-Fuchs,Peter Kapeller,Helena Schmidt,Birgit Poltrum,Reinhold E. Schmidt,Hans-Peter Hartung,Franz Fazekas +8 more
TL;DR: The ϵ4 allele of apolipoprotein E (APOEϵ4) has a negative effect on the course of MS, and increasing axonal damage may be an important mechanism.