G
Geoffrey M. Cooper
Researcher at Boston University
Publications - 174
Citations - 20081
Geoffrey M. Cooper is an academic researcher from Boston University. The author has contributed to research in topics: Gene & Transfection. The author has an hindex of 56, co-authored 174 publications receiving 19698 citations. Previous affiliations of Geoffrey M. Cooper include Harvard University & University of Washington.
Papers
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Journal ArticleDOI
The c-mos gene product is required for cyclin B accumulation during meiosis of mouse eggs.
TL;DR: The c-mos protooncogene appears to affect the pathway that regulates cyclin B stability during meiosis of mouse eggs, which is correlated with a failure of the injected eggs to accumulate newly synthesizedcyclin B.
Book
Elements of Human Cancer
TL;DR: This best-selling volume provides a broad overview of cancer from the basic biology and causes of human cancer through detailed discussion of the major types of cancer.
Journal ArticleDOI
Glycogen synthase kinase-3 represses cyclic AMP response element-binding protein (CREB)-targeted immediate early genes in quiescent cells.
TL;DR: The results indicate that GSK-3 actively represses gene expression in quiescent cells, with inhibition of CREB playing a key role in this transcriptional response.
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Transformation of NIH/3T3 mouse cells by DNA of Rous sarcoma virus
TL;DR: It appeared that transformation of NIH/3T3 cells by RSV DNA occurred by direct integration and stable expression of the donor DNA, rather than by production of extracellular progeny virus and secondary virus infection, as was previously shown to be required for transformation of chicken embryo fibroblasts byRSV DNA.
Journal ArticleDOI
Hydrolysis of phosphatidylcholine is stimulated by Ras proteins during mitogenic signal transduction.
Hong Cai,Peter Erhardt,József Szeberényi,Maria T. Diaz-Meco,Terje Johansen,Jorge Moscat,Geoffrey M. Cooper +6 more
TL;DR: PC hydrolysis is a target of Ras during the transduction of growth factor-initiated mitogenic signals, suggesting that PC-derived second messengers may act downstream of Ras in mitogenic signal transduction.