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Gesa Hartwigsen

Researcher at Max Planck Society

Publications -  151
Citations -  4229

Gesa Hartwigsen is an academic researcher from Max Planck Society. The author has contributed to research in topics: Transcranial magnetic stimulation & Medicine. The author has an hindex of 28, co-authored 112 publications receiving 2935 citations. Previous affiliations of Gesa Hartwigsen include University of Hamburg & University of Kiel.

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How does transcranial magnetic stimulation modify neuronal activity in the brain? Implications for studies of cognition

TL;DR: In this paper, it is shown that the response to TMS depends on how excitable the cortex is at the time the stimulus is applied: if many neurones are close to firing threshold then the more of them are recruited by the pulse than at rest.
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Combining non-invasive transcranial brain stimulation with neuroimaging and electrophysiology: Current approaches and future perspectives

TL;DR: A conceptual framework is provided, emphasizing principal strategies and highlighting promising future directions to exploit the benefits of combining NTBS with neuroimaging or electrophysiology to close the loop between measuring and modulating brain activity by means of closed-loop brain state-dependent NTBS.

How does transcranial magnetic stimulation modify neuronal activity in the brain? Implications for studies of cognition

TL;DR: An important feature of the response to TMS is "context dependency", which indicates that the response depends on how excitable the cortex is at the time the stimulus is applied: if many neurones are close to firing threshold then the more of them are recruited by the pulse than at rest.
Journal ArticleDOI

Damage to ventral and dorsal language pathways in acute aphasia.

TL;DR: The results from patients with acute stroke lesions support the claim that language is organized along two segregated dorsal–ventral streams, and is the first lesion study demonstrating that task performance on auditory comprehension measures requires an interaction between temporal and prefrontal brain regions via the ventral extreme capsule pathway.