G
Giovanni Barosi
Researcher at University of Pavia
Publications - 352
Citations - 21433
Giovanni Barosi is an academic researcher from University of Pavia. The author has contributed to research in topics: Myelofibrosis & Essential thrombocythemia. The author has an hindex of 69, co-authored 334 publications receiving 19225 citations.
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Journal ArticleDOI
Clinical and economic impact of epoetins in cancer care.
Monia Marchetti,Giovanni Barosi +1 more
TL;DR: In the near future, it is expected that a wider range of epoetins, drug patent expiry, a more appropriate patient selection criteria and an improved dosage schedule may help increase the efficiency of cancer-related anaemia management.
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Testing for occult cancer in patients with idiopathic deep vein thrombosis : A decision analysis
TL;DR: According to the effectiveness criterion adopted, the only worthwhile investigation strategy includes colon and breast cancer in females and testing for colon cancer in males is desirable at a lower criterion of effectiveness.
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Classification of anaemia on the basis of ferrokinetic parameters
TL;DR: The present functional classification of anaemia provides a complete picture of the different pathogenetic mechanisms and may represent the basis for a more rational diagnostic approach to erythroid disorders.
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Primary myelofibrosis: Older age and high JAK2V617F allele burden are associated with elevated plasma high-sensitivity C-reactive protein levels and a phenotype of progressive disease.
Giovanni Barosi,Margherita Massa,Rita Campanelli,Gabriela Fois,Paolo Catarsi,Gianluca Viarengo,Laura Villani,Valentina Poletto,Tiziana Bosoni,Umberto Magrini,Robert Peter Gale,Vittorio Rosti +11 more
TL;DR: The data indicate that older age and high JAK2V617 allele burden are major determinants of inflammation in PMF, and are associated with disease progression.
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Studies of the site and distribution of CD34+ cells in idiopathic myelofibrosis
TL;DR: The findings suggest that the BM fibrosis and abnormal hematopoietic stem cell distribution in patients with IM is a consequence of the progeny of a malignant hematoplastic stem cell clone.