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Greg M. Murray

Researcher at University of Sydney

Publications -  127
Citations -  8180

Greg M. Murray is an academic researcher from University of Sydney. The author has contributed to research in topics: Lateral pterygoid muscle & Orofacial pain. The author has an hindex of 42, co-authored 126 publications receiving 7001 citations. Previous affiliations of Greg M. Murray include Westmead Hospital & University of Toronto.

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Diagnostic Criteria for Temporomandibular Disorders (DC/TMD) for Clinical and Research Applications: recommendations of the International RDC/TMD Consortium Network* and Orofacial Pain Special Interest Group†

TL;DR: The newly recommended evidence-based new DC/TMD protocol is appropriate for use in both clinical and research settings and includes both a valid screener for detecting any pain-related TMD as well as valid diagnostic criteria for differentiating the most common pain- related TMD.
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Different Pain, Different Brain: Thalamic Anatomy in Neuropathic and Non-Neuropathic Chronic Pain Syndromes

TL;DR: In trigeminal neuropathy patients, magnetic resonance spectroscopy revealed a significant reduction in the N-acetylaspartate/creatine ratio, a biochemical marker of neural viability, in the region of thalamic volume loss, suggesting that neuropathic pain conditions that result from peripheral injuries may be generated and/or maintained by structural changes in regions such as the thalamus.
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Organization of the primate face motor cortex as revealed by intracortical microstimulation and electrophysiological identification of afferent inputs and corticobulbar projections.

TL;DR: Clear evidence for multiple representation of a particular muscle is found, thus supporting other investigations of the motor cortex, which indicate that multiple, yet discrete, efferent microzones represent an essential organizational principle of theMotor cortex.
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Chronic Pain: Lost Inhibition?

TL;DR: The data suggest that chronic neuropathic pain is associated with altered thalamic anatomy and activity, which may result in disturbed thalamocortical circuits, which could result in the constant perception of pain.