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Györgyi Csankovszki

Researcher at University of Michigan

Publications -  34
Citations -  2723

Györgyi Csankovszki is an academic researcher from University of Michigan. The author has contributed to research in topics: Dosage compensation & Dosage compensation complex. The author has an hindex of 18, co-authored 33 publications receiving 2514 citations. Previous affiliations of Györgyi Csankovszki include Massachusetts Institute of Technology.

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Loss of genomic methylation causes p53-dependent apoptosis and epigenetic deregulation.

TL;DR: The results demonstrate that loss of Dnmt1 causes cell-type–specific changes in gene expression that impinge on several pathways, including expression of imprinted genes, cell-cycle control, growth factor/receptor signal transduction and mobilization of retroelements.
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Synergism of Xist Rna, DNA Methylation, and Histone Hypoacetylation in Maintaining X Chromosome Inactivation

TL;DR: Using a conditional mutant Xist allele, direct evidence is provided for that loss of Xist RNA destabilizes the inactive state in somatic cells, leading to an increased reactivation frequency of an X-linked GFP transgene and of the endogenous hypoxanthine phosphoribosyl transferase (Hprt) gene in mouse embryonic fibroblasts.
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Conditional deletion of Xist disrupts histone macroH2A localization but not maintenance of X inactivation.

TL;DR: Conditional deletion of Xist disrupts histone macroH2A localization but not maintenance of X inactivation; however, this does not affect the ability of X to be reprogramed for inactivation.
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Three Distinct Condensin Complexes Control C. elegans Chromosome Dynamics

TL;DR: It is demonstrated that condensin subunits in C. elegans form three complexes, one that functions in dosage compensation and two that function in mitosis and meiosis, illustrating the versatility of condensins to function in both gene regulation and chromosome segregation.