H
Hiroyuki Tanaka
Researcher at Hokkaido University
Publications - 112
Citations - 2150
Hiroyuki Tanaka is an academic researcher from Hokkaido University. The author has contributed to research in topics: Consolidation (soil) & Troponin C. The author has an hindex of 24, co-authored 112 publications receiving 1932 citations. Previous affiliations of Hiroyuki Tanaka include Ehime University.
Papers
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Detoxification mechanism of heavy metals in marine mammals and seabirds: interaction of selenium with mercury, silver, copper, zinc, and cadmium in liver.
Tokutaka Ikemoto,Takashi Kunito,Hiroyuki Tanaka,Norihisa Baba,Nobuyuki Miyazaki,Shinsuke Tanabe +5 more
TL;DR: It is notable that the existing state of silver was similar to that of mercury as judged by their subcellular distribution and the extraction tests, suggesting that silver also interacted with selenium in the liver of marine animals used in this study.
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Influences of diatom microfossils on engineering properties of soils
TL;DR: In this article, the authors examined how geotechnical characteristics of soils can be influenced by the presence of diatom microfossils, using an artificially prepared mixture of soil and diatomite.
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Global pollution monitoring of PCBs and organochlorine pesticides using skipjack tuna as a bioindicator.
D. Ueno,Shin Takahashi,Hiroyuki Tanaka,Annamalai Subramanian,Gilberto Fillmann,Haruhiko Nakata,Paul K.S. Lam,J. Zheng,Muswerry Muchtar,Maricar S. Prudente,K. H. Chung,Shinsuke Tanabe +11 more
TL;DR: In this paper, the liver of skipjack tuna (Katsuwonus pelamis) collected from the offshore waters of various regions in the world (offshore waters around Japan, Taiwan, Philippines, Indonesia, Seychelles, and Brazil, and the Japan Sea, the East China Sea, South China sea, the Bay of Bengal, and North Pacific Ocean).
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Isolation and primary structure of a cellulase from the Japanese sea urchin Strongylocentrotus nudus.
TL;DR: It is confirmed that the sea urchin produces an active GHF9 cellulase closely related to other animal cellulases.
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Drastic Ca2+ sensitization of myofilament associated with a small structural change in troponin I in inherited restrictive cardiomyopathy.
Fumiaki Yumoto,Qunwei Lu,Sachio Morimoto,Hiroyuki Tanaka,Naoko Kono,Koji Nagata,Takao Ojima,Fumi Takahashi-Yanaga,Yoshikazu Miwa,Toshiyuki Sasaguri,Kiyoyoshi Nishita,Masaru Tanokura,Iwao Ohtsuki +12 more
TL;DR: It is suggested that both HCM and RCM involving cTnI mutations share a common feature of increased Ca2+ sensitivity of cardiac myofilament, but more severe change in Ca2-sensitizing effect is associated with the clinical phenotype of RCM.