Hongbing Xu

TL;DR: Assessment of variations, sources, and lifetime excessive cancer risk (ECR) attributable to PAHs bound to ambient particulate matters with aerodynamic diameter less than 2.5μm in metropolitan Beijing, China suggested that ambient particulates reduction from energy reconstruction and adaption of clean fuels would result in reductions PM2.5-boundPAHs and its associated cancer risks.

TL;DR: Higher ambient air pollution concentrations were associated with impairments in HDL functionality, potentially because of systemic inflammation and oxidative stress, and these novel findings further the understanding of the mechanisms whereby air pollutants promote cardiometabolic disorders.

TL;DR: It was showed that indoor air filtration produced clear improvement on indoor air quality, but no demonstrable changes in the cardio-respiratory outcomes of study interest observed in the seniors living with real-world air pollution exposures.

TL;DR: In this article, the pathophysiologic mechanisms of air pollution-associated exacerbation of cardiovascular events remain incompletely understood, and the authors assess whether ambient air pollution can cause cardiovascular events.

TL;DR: It is demonstrated that short-term exposures to ambient PM2.5 in a highly polluted environment can promote elevations in BP even among healthy adults, and the fact that no adverse hemodynamic responses were observed in a clean location supports the key public health importance of international efforts to improve air quality as part of the global battle against hypertension.