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Ingrid Dieset

Researcher at Oslo University Hospital

Publications -  60
Citations -  2311

Ingrid Dieset is an academic researcher from Oslo University Hospital. The author has contributed to research in topics: Schizophrenia & Bipolar disorder. The author has an hindex of 23, co-authored 56 publications receiving 1774 citations. Previous affiliations of Ingrid Dieset include University of Oslo.

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Increased mortality in schizophrenia due to cardiovascular disease - a non-systematic review of epidemiology, possible causes, and interventions.

TL;DR: There is an urgent need to develop and implement effective programs to increase life expectancy in schizophrenia, and it is argued that mental health workers should be more involved in this important task.
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Oxytocin pathway gene networks in the human brain

TL;DR: In this paper, the authors characterized the distribution of OXT, OXTR, and CD38 mRNA across the human brain by creating voxel-by-voxel volumetric expression maps, and identified putative gene pathway interactions by comparing gene expression patterns across 20,737 genes.
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Childhood Trauma Is Associated With Severe Clinical Characteristics of Bipolar Disorders

TL;DR: The results demonstrate consistent associations between childhood trauma and more severe clinical characteristics in bipolar disorder and show the importance of including emotional abuse as well as the more frequently investigated sexual abuse when targeting clinical characteristics of bipolar disorder.
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Affective symptoms are associated with markers of inflammation and immune activation in bipolar disorders but not in schizophrenia

TL;DR: The current associations between inflammatory markers and affective symptomatology in BP and not SCZ suggest that immune related mechanisms are associated with core psychopathology of BP.
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Interleukin 1 receptor antagonist and soluble tumor necrosis factor receptor 1 are associated with general severity and psychotic symptoms in schizophrenia and bipolar disorder.

TL;DR: It is found that inflammatory markers, particularly IL-1Ra and sTNF-R1 are associated with both general disease severity and psychotic features, which supports a role of immune activation in the core pathological mechanisms of severe mental disorders.