J
Jason B. Harris
Researcher at Harvard University
Publications - 202
Citations - 9092
Jason B. Harris is an academic researcher from Harvard University. The author has contributed to research in topics: Cholera & Vibrio cholerae. The author has an hindex of 42, co-authored 185 publications receiving 7571 citations. Previous affiliations of Jason B. Harris include International Centre for Diarrhoeal Disease Research, Bangladesh & University of Tennessee.
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Journal ArticleDOI
Characterization of dystrophin in muscle-biopsy specimens from patients with Duchenne's or Becker's muscular dystrophy
Eric P. Hoffman,Fischbeck Kh,Robert H. Brown,Mark D. Johnson,Medori R,John D. Loike,Jason B. Harris,R Waterston,Brooke M,Linda A. Specht +9 more
TL;DR: These data show the clinical consequences of both quantitative alterations (in Duchenne's and intermediate dystrophy) in a single protein, and the biochemical assay for dystrophin should prove helpful in delineating myopathies that overlap clinically with DuchenNE's and Becker's Dystrophies and shows promise as an accurate diagnostic tool.
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The origin of the Haitian cholera outbreak strain.
Chen-Shan Chin,Jon M. Sorenson,Jason B. Harris,William P. Robins,Richelle C. Charles,Roger R. Jean-Charles,James H. Bullard,Dale R. Webster,Andrew Kasarskis,Paul Peluso,Ellen E. Paxinos,Yoshiharu Yamaichi,Stephen B. Calderwood,John J. Mekalanos,Eric E. Schadt,Matthew K. Waldor,Matthew K. Waldor +16 more
TL;DR: The Haitian epidemic is probably the result of the introduction, through human activity, of a V. cholerae strain from a distant geographic source, and analysis of genomic variation of the Haitian isolates reveals a more distant relationship with circulating South American isolates.
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Cholera transmission: the host, pathogen and bacteriophage dynamic
TL;DR: Advances that will help to unravel how interactions between the host, the bacterial pathogen and the lytic bacteriophage might propel and quench cholera outbreaks in endemic settings and in emergent epidemic regions such as Zimbabwe are highlighted.
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Persistence and decay of human antibody responses to the receptor binding domain of SARS-CoV-2 spike protein in COVID-19 patients.
Anita S. Iyer,Forrest K. Jones,Ariana Nodoushani,Meagan Kelly,Margaret Becker,Damien Slater,Rachel Mills,Erica Teng,Mohammad Kamruzzaman,Wilfredo F. Garcia-Beltran,Michael G Astudillo,Diane Yang,Tyler E. Miller,Elizabeth Oliver,Stephanie Fischinger,Caroline Atyeo,A. John Iafrate,Stephen B. Calderwood,Stephen A. Lauer,Jingyou Yu,Zhenfeng Li,Jared Feldman,Blake M. Hauser,Timothy M. Caradonna,John A. Branda,Sarah E Turbett,Regina C. LaRocque,Guillaume Mellon,Dan H. Barouch,Dan H. Barouch,Aaron G. Schmidt,Aaron G. Schmidt,Andrew S. Azman,Galit Alter,Edward T. Ryan,Jason B. Harris,Richelle C. Charles +36 more
TL;DR: RBD-targeted antibodies are excellent markers of previous and recent infection, that differential isotype measurements can help distinguish between recent and older infections, and that IgG responses persist over the first few months after infection and are highly correlated with neutralizing antibodies.
Journal ArticleDOI
Improved diagnosis of Becker muscular dystrophy by dystrophin testing.
TL;DR: Dystrophin analysis is required for accurately distinguishing between Becker dystrophy and clinically similar autosomal recessive myopathies, and the correlation of both the biochemical and clinical data suggests that Duchenne/Becker Dystrophy can be divided into 4 clinically useful categories.