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Jean-Christophe Lagier

Researcher at Aix-Marseille University

Publications -  311
Citations -  17625

Jean-Christophe Lagier is an academic researcher from Aix-Marseille University. The author has contributed to research in topics: Genome & Medicine. The author has an hindex of 50, co-authored 274 publications receiving 13861 citations. Previous affiliations of Jean-Christophe Lagier include Institut de recherche pour le développement & Centre national de la recherche scientifique.

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Akkermansia muciniphila: The state of the art, 18 years after its first discovery

TL;DR: An overview of what is currently known about A. muciniphila’s phenotypical and genotypic traits, as well as its culture techniques and its connections to a number of human diseases and its potential application as an effective next generation probiotic is provided.
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Acute polyarthritis in a young patient caused by meningococcal and parvovirus B19 infections: a case report and review of the literature.

TL;DR: The first case report of polyarthritis caused by concomitant meningococcal and parvovirus B19 infections in a young adult is reported, illustrating the need for a systematic approach to the diagnosis of febrile acutepolyarthritis.
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Sequencing of Monkeypox virus from infected patients reveals viral genomes with APOBEC3-like editing, gene inactivation, and bacterial agents of skin superinfection

TL;DR: DNA metagenomic analyses of clinical samples from MPX virus-infected patients between June and July 2022, using next-generation sequencing with Illumina or Nanopore technologies point out the common presence in monkeypox lesions of bacterial agents of skin superinfection, which warrants a close clinical monitoring inmonkeypox patients.
Posted ContentDOI

The emergence, spread and vanishing of a French SARS-CoV-2 variant exemplifies the fate of RNA virus epidemics and obeys the Black Queen rule

TL;DR: It is speculated that the observed heterogenicity of a lineage may predict the end of the outbreak of SARS-CoV-2, because several beneficial mutations at unpredicted sites initiated a new outbreak, while the accumulation of other mutations resulted in more viralheterogenicity, increased diversity and vanishing of the lineages.