J
Jeffrey V. Ravetch
Researcher at Rockefeller University
Publications - 310
Citations - 59480
Jeffrey V. Ravetch is an academic researcher from Rockefeller University. The author has contributed to research in topics: Antibody & Receptor. The author has an hindex of 110, co-authored 296 publications receiving 54829 citations. Previous affiliations of Jeffrey V. Ravetch include Bristol-Myers Squibb & Kettering University.
Papers
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Journal ArticleDOI
Functional diversification of IgGs through Fc glycosylation
Taia T. Wang,Jeffrey V. Ravetch +1 more
TL;DR: How Fc structures arising from sialylation and fucosylation impact immunity is discussed, focusing on responses to vaccination and infection.
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Reconstitution of human Fc gamma RIII cell type specificity in transgenic mice.
TL;DR: These studies define the elements conferring the cell type-specific expression of the human Fc gamma RIII genes within the 5' flanking sequences and first intron of thehuman Fc Gamma RIIIA and FcGamma RIIIB genes.
Journal ArticleDOI
Intravenous immune globulin prevents venular vaso-occlusion in sickle cell mice by inhibiting leukocyte adhesion and the interactions between sickle erythrocytes and adherent leukocytes.
Aslihan Turhan,Pegah Jenab,Pierre Bruhns,Jeffrey V. Ravetch,Barry S. Coller,Paul S. Frenette +5 more
TL;DR: The effects of commercial intravenous human immune globulin (i.v.IG) preparations are studied and it is raised the possibility that i.V.IG may have a beneficial effect on sickle cell-associated vaso-occlusion.
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Interferon-inducible gene maps to a chromosomal band associated with a (4;11) translocation in acute leukemia cells.
TL;DR: The results suggest a model in which juxtaposition of genetic loci regulated by antiproliferative signals, such as interferon, next to an oncogene, like ETS1, could effectively short circuit homeostatic control circuits and contribute to the neoplastic state.
Journal Article
Cutting Edge: Role of the Inositol Phosphatase SHIP in B Cell Receptor-Induced Ca2+ Oscillatory Response
Hidetaka Okada,Silvia Bolland,Akiko Hashimoto,Mari Kurosaki,Yukihito Kabuyama,Masamitsu Iino,Jeffrey V. Ravetch,Tomohiro Kurosaki +7 more
TL;DR: It is reported that SHIP-deficient DT40 B cells display enhanced Ca2+ mobilization in response to BCR ligation, whereas extracellular signal-regulated kinase activation is unaffected.