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Jing Liu

Researcher at China Medical University (PRC)

Publications -  88
Citations -  2429

Jing Liu is an academic researcher from China Medical University (PRC). The author has contributed to research in topics: Cancer cell & Apoptosis. The author has an hindex of 26, co-authored 80 publications receiving 1997 citations.

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Knockdown of long non-coding RNA XIST exerts tumor-suppressive functions in human glioblastoma stem cells by up-regulating miR-152

TL;DR: It is proved that XIST expression was up-regulated in glioma tissues and GSCs and miR-152 mediated the tumor-suppressive effects that knockdown of XIST exerted.
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β-Elemene-induced autophagy protects human gastric cancer cells from undergoing apoptosis

TL;DR: The authors' data provides the first evidence that β-elemene induces protective autophagy and prevents human gastric cancer cells from undergoing apoptosis, and a combination of β- elemene with autophagic inhibitor might thus be a useful therapeutic option for advanced Gastric cancer.
Journal Article

Long non-coding RNA MALAT-1 is downregulated in preeclampsia and regulates proliferation, apoptosis, migration and invasion of JEG-3 trophoblast cells.

TL;DR: The results suggest that MALAT-1 may play an important role in the regulation of proliferation, cell cycle, apoptosis, migration and invasion of trophoblast cells, and under-expression of MALat-1 during early placentation may be involved in the pathogenesis of preeclampsia.
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Arsenic trioxide induces apoptosis and G2/M phase arrest by inducing Cbl to inhibit PI3K/Akt signaling and thereby regulate p53 activation.

TL;DR: It is demonstrated that inhibition of PI3K/Akt signaling by Cbl is involved in both ATO-induced apoptosis of NB4 cells and ATo-induced G2/M phase arrest of gastric cancer cells.
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KIR3DS1/L1 and HLA-Bw4-80I are associated with HIV disease progression among HIV typical progressors and long-term nonprogressors

TL;DR: The authors' data suggest that different KIR-HLA genotypes and different levels of transcripts associate with HIV disease progression, and this study confirmed that homozygosity for the HLA-Bw6 allele was associated with rapid disease progression.