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John Schiefelbein

Researcher at University of Michigan

Publications -  106
Citations -  11230

John Schiefelbein is an academic researcher from University of Michigan. The author has contributed to research in topics: Arabidopsis & Root hair. The author has an hindex of 50, co-authored 102 publications receiving 10289 citations. Previous affiliations of John Schiefelbein include Michigan State University.

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Cell Identity Mediates the Response of Arabidopsis Roots to Abiotic Stress

TL;DR: The transcriptional response to high salinity of different cell layers and developmental stages of the Arabidopsis root is characterized and it is found that transcriptional responses are highly constrained by developmental parameters.
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WEREWOLF, a MYB-Related Protein in Arabidopsis, Is a Position-Dependent Regulator of Epidermal Cell Patterning

TL;DR: A novel gene, WEREWOLF (WER), is described here that is required for position-dependent patterning of the epidermal cell types of Arabidopsis and is shown to regulate the position- dependent expression of the GLABRA2 homeobox gene, to interact with a bHLH protein, and to act in opposition to the CAPRICE MYB.
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The TTG Gene Is Required to Specify Epidermal Cell Fate and Cell Patterning in the Arabidopsis Root

TL;DR: The results indicate that alterations in TTG activity cause developing epidermal cells to misinterpret their position and differentiate into inappropriate cell types, suggesting that, in wild-type roots, TTG provides, or responds to, positional signals to cause differentiating epidersmal cells that lie over cortical cells to adopt a hairless cell fate.
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The homeobox gene GLABRA2 is required for position-dependent cell differentiation in the root epidermis of Arabidopsis thaliana

TL;DR: Results indicate that the GL2 homeodomain protein normally regulates a subset of the processes that occur during the differentiation of hairless epidermal cells of the Arabidopsis root, and appears to act in a cell-position-dependent manner to suppress hair formation in differentiating hairless cells.
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Mutant of Arabidopsis Deficient in Xylem Loading of Phosphate

TL;DR: The results suggest that the mutant is deficient in activity of a protein required to load phosphate into the xylem, suggesting that the phosphate deficiency is caused by a single nuclear recessive mutation at a locus designated pho1.