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John W. Erdman

Researcher at University of Illinois at Urbana–Champaign

Publications -  329
Citations -  19074

John W. Erdman is an academic researcher from University of Illinois at Urbana–Champaign. The author has contributed to research in topics: Lycopene & Carotenoid. The author has an hindex of 65, co-authored 314 publications receiving 17580 citations. Previous affiliations of John W. Erdman include University of Oklahoma Health Sciences Center & Urbana University.

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Tissue Stores of β-Carotene Are Not Conserved for Later Use as a Source of Vitamin A during Compromised Vitamin A Status in Mongolian Gerbils (Meriones unguiculatus)

TL;DR: The results from both studies suggest that a substantial amount of hepatic betaC is rapidly lost when BetaC is eliminated from the diet and therefore is not conserved to meet later VA needs.
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Liver Fat Assessment in Multiview Sonography Using Transfer Learning With Convolutional Neural Networks

TL;DR: In this article, the authors developed and evaluated deep learning models devised for liver fat assessment based on ultrasound (US) images acquired from four different liver views: transverse plane (hepatic veins at the confluence with the inferior vena cava, right portal vein, right posterior portal vein) and sagittal plane (liver/kidney).
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Low-lycopene containing tomato powder diet does not protect against prostate cancer in TRAMP mice

TL;DR: A low-lycopene TP intervention failed to reduce carcinogenesis in TRAMP mice, suggesting a delay in the progression of prostate cancer.
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Intracellular β-carotene transport in bovine liver and intestine is not mediated by cytosolic proteins

TL;DR: The results suggest that unlike many other lipid species, intracellular transport of beta-carotene is not mediated by cytosolic transport proteins and must occur by other mechanisms such as vesicular transport or by membrane-bound proteins.
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Vitamin A induced hypertriglyceridemia in cholesterol-fed rats.

TL;DR: Results show that daily supplement of vitamin A increased serum triglycerides and reduced serum and hepatic cholesterol concentrations, and Serum and liver alterations were dependent on continued feeding.