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Joyce S. Balami

Researcher at University of Oxford

Publications -  37
Citations -  1885

Joyce S. Balami is an academic researcher from University of Oxford. The author has contributed to research in topics: Stroke & Thrombolysis. The author has an hindex of 15, co-authored 35 publications receiving 1534 citations. Previous affiliations of Joyce S. Balami include John Radcliffe Hospital & Norfolk and Norwich University Hospitals NHS Foundation Trust.

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Journal ArticleDOI

Complications of intracerebral haemorrhage.

TL;DR: Improved surveillance is needed for the prevention of associated complications of ICH, or, when this is not possible, early detection and optimum management, which could be effective in the reduction of adverse effects early in the course of stroke and in the improvement of prognosis.
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Neuroprotection for ischaemic stroke: Translation from the bench to the bedside

TL;DR: A number of promising neuroprotectants in preclinical development including haematopoietic growth factors, and inhibitors of the nicotinamide adenine dinucleotide phosphate oxidases, a source of free radical production which is a key step in the pathophysiology of acute ischaemic stroke.
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Neurological complications of acute ischaemic stroke.

TL;DR: There is a clear need for improved surveillance and specific interventions for the prevention, early diagnosis, and proper management of neurological complications during the acute phase of stroke to reduce stroke morbidity and mortality.
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Ischemic stroke in the elderly: an overview of evidence

TL;DR: Future research on ischemic stroke should consider age as a factor that influences stroke prevention and treatment, and should focus on the management of acute stroke in the elderly to reduce the incidence and improve outcomes in this vulnerable group.
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Effects of fluoxetine on functional outcomes after acute stroke (FOCUS): a pragmatic, double-blind, randomised, controlled trial

Martin Dennis, +1271 more
- 19 Jan 2019 - 
TL;DR: Although the treatment reduced the occurrence of depression, it increased the frequency of bone fractures and these results do not support the routine use of fluoxetine either for the prevention of post-stroke depression or to promote recovery of function.