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Neil Hunter

Researcher at Howard Hughes Medical Institute

Publications -  71
Citations -  8949

Neil Hunter is an academic researcher from Howard Hughes Medical Institute. The author has contributed to research in topics: Homologous recombination & Meiosis. The author has an hindex of 36, co-authored 67 publications receiving 7646 citations. Previous affiliations of Neil Hunter include University of California & University of California, Berkeley.

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Neurological and neuropsychiatric complications of COVID-19 in 153 patients: a UK-wide surveillance study.

Aravinthan Varatharaj, +125 more
TL;DR: This is the first nationwide, cross-specialty surveillance study of acute neurological and psychiatric complications of COVID-19 and provides valuable and timely data that are urgently needed by clinicians, researchers, and funders.
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The Single-End Invasion: An Asymmetric Intermediate at the Double-Strand Break to Double-Holliday Junction Transition of Meiotic Recombination

TL;DR: It is shown that the structural asymmetry of SEIs indicates that the two ends of a DSB interact with the homolog in temporal succession, via structurally (and thus biochemically) distinct processes, which can explain crossover suppression between homeologous and structurally heterozygous chromosomes.
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Crossover/noncrossover differentiation, synaptonemal complex formation, and regulatory surveillance at the leptotene/zygotene transition of meiosis.

TL;DR: During wild-type meiosis, recombinational interactions are differentiated into CR and NCR types very early, prior to onset of stable strand exchange and independent of SC, suggesting that SC formation may require interference.
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Meiotic Recombination: The Essence of Heredity

TL;DR: This review highlights the features of meiotic recombination that distinguish it from recombinational repair in somatic cells, and how the molecular processes of meiotics recombination are embedded and interdependent with the chromosome structures that characterize meiotic prophase.
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The Mus81/Mms4 endonuclease acts independently of double-Holliday junction resolution to promote a distinct subset of crossovers during meiosis in budding yeast.

TL;DR: This work provides three lines of evidence that Mus81/Mms4 is not the major meiotic HJ resolvase in S. cerevisiae and reveals the existence of two distinct classes of crossovers in budding yeast.