J
Juan Jose Martin
Researcher at University of Utah
Publications - 12
Citations - 743
Juan Jose Martin is an academic researcher from University of Utah. The author has contributed to research in topics: Cervical Artery & Stroke. The author has an hindex of 11, co-authored 12 publications receiving 586 citations. Previous affiliations of Juan Jose Martin include Heidelberg University & National University of Cordoba.
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Journal ArticleDOI
Epidemiology, pathophysiology, diagnosis, and management of intracranial artery dissection
Stéphanie Debette,Stéphanie Debette,Annette Compter,M.-A. Labeyrie,Maarten Uyttenboogaart,T. M. Metso,Jennifer J. Majersik,Barbara Goeggel-Simonetti,Stefan T. Engelter,Alessandro Pezzini,Philippe Bijlenga,Andrew M. Southerland,Olivier Naggara,Yannick Béjot,John W. Cole,Anne Ducros,Giacomo Giacalone,Sabrina Schilling,Peggy Reiner,Hakan Sarikaya,Janna C Welleweerd,L. Jaap Kappelle,Gert J. de Borst,Leo H. Bonati,Simon Jung,Vincent Thijs,Juan Jose Martin,Tobias Brandt,Caspar Grond-Ginsbach,Manja Kloss,Tohru Mizutani,Kazuo Minematsu,James F. Meschia,Vitor Mendes Pereira,Anna Bersano,Emmanuel Touzé,Philippe Lyrer,Didier Leys,Hugues Chabriat,Hugh S. Markus,Bradford B. Worrall,Stéphane Chabrier,R W Baumgartner,Christian Stapf,Turgut Tatlisumak,Marcel Arnold,Marie-Germaine Bousser +46 more
TL;DR: Most patients with subarachnoid haemorrhage undergo surgical or endovascular treatment to prevent rebleeding, whereas patients with intracranial artery dissection and cerebral ischaemia are treated with antithrombotics.
Journal ArticleDOI
Cervical artery dissection Trauma and other potential mechanical trigger events
Stefan T. Engelter,Caspar Grond-Ginsbach,Tiina M. Metso,Antti J. Metso,Manja Kloss,Stéphanie Debette,Didier Leys,Armin J. Grau,Jean Dallongeville,Marie Bodenant,Yves Samson,Valeria Caso,Alessandro Pezzini,Leo H. Bonati,Vincent Thijs,Henrik Gensicke,Juan Jose Martin,Anna Bersano,Emmanuel Touzé,Turgut Tatlisumak,Philippe Lyrer,Tobias Brandt +21 more
TL;DR: CeAD patients with PCT were younger and presented more often with neck pain and less often with stroke than CeAD patients without PCT, but was not an independent outcome predictor.
Journal ArticleDOI
Familial occurrence and heritable connective tissue disorders in cervical artery dissection
Stéphanie Debette,Barbara Goeggel Simonetti,Sabrina Schilling,Juan Jose Martin,Manja Kloss,Hakan Sarikaya,Hakan Sarikaya,Ingrid Hausser,Stefan T. Engelter,Tiina M. Metso,Alessandro Pezzini,Vincent Thijs,Emmanuel Touzé,Emmanuel Touzé,Stefano Paolucci,Paolo Costa,Maria Sessa,Yves Samson,Yannick Béjot,Ayse Altintas,Antti J. Metso,Dominique Hervé,Dominique Hervé,Christoph Lichy,Simon Jung,Urs Fischer,C. Lamy,Armin J. Grau,Hugues Chabriat,Hugues Chabriat,Valeria Caso,Philippe Lyrer,Christian Stapf,Turgut Tatlisumak,Tobias Brandt,Elisabeth Tournier-Lasserve,Elisabeth Tournier-Lasserve,Dominique P. Germain,Michael Frank,Ralf W. Baumgartner,Caspar Grond-Ginsbach,Marie-Germaine Bousser,Didier Leys,Jean Dallongeville,Anna Bersano,Marcel Arnold +45 more
TL;DR: In this largest series of patients with CeAD to date, family history of symptomatic CeAD was rare and inherited connective tissue disorders seemed exceptional, supporting the notion that CeAD is a multifactorial disease in the vast majority of cases.
Journal ArticleDOI
Familial Cervical Artery Dissections Clinical, Morphologic, and Genetic Studies
Juan Jose Martin,Ingrid Hausser,Philippe Lyrer,Otto Busse,Ralf Schwarz,R. Schneider,Tobias Brandt,Manja Kloss,Markus Schwaninger,Stefan T. Engelter,Caspar Grond-Ginsbach +10 more
TL;DR: Familial CAD patients are young and probably are at high risk for recurrent or multiple CAD, and the finding of a COL3A1 mutation revealed the presence of an inherited connective tissue disorder in 1 family.
Journal ArticleDOI
Clinical import of Horner syndrome in internal carotid and vertebral artery dissection
Philippe Lyrer,Tobias Brandt,Tiina M. Metso,Antti J. Metso,Manja Kloss,Stéphanie Debette,Didier Leys,Valeria Caso,Alessandro Pezzini,Leo H. Bonati,Vincent Thijs,Anna Bersano,Emmanuel Touzé,Henrik Gensicke,Juan Jose Martin,Christoph Lichy,Turgut Tatlisumak,Stefan T. Engelter,Caspar Grond-Ginsbach +18 more
TL;DR: In patients with ICAD, HS is an easily assessable marker that might indicate a more benign clinical course and HS had no prognostic meaning in patients with VAD.