J
Juan Wang
Researcher at Fifth Affiliated Hospital of Xinjiang Medical University
Publications - 4
Citations - 96
Juan Wang is an academic researcher from Fifth Affiliated Hospital of Xinjiang Medical University. The author has contributed to research in topics: Gene knockdown & Pressure overload. The author has an hindex of 3, co-authored 4 publications receiving 45 citations.
Papers
More filters
Journal ArticleDOI
Andrographolide Protects Against Adverse Cardiac Remodeling After Myocardial Infarction through Enhancing Nrf2 Signaling Pathway.
Saiyang Xie,Wei Deng,Jiao-Jiao Chen,Qing-Qing Wu,Hongjian Li,Juan Wang,Li Wei,Chen Liu,Mingxia Duan,Zhulan Cai,Qingwen Xie,Tongtong Hu,Xiao-Feng Zeng,Qi-Zhu Tang +13 more
TL;DR: It is suggested that Andr alleviates adverse cardiac remodeling following myocardial infarction through enhancing Nrf2 signaling pathway.
Journal ArticleDOI
Long non-coding RNA cytoskeleton regulator RNA (CYTOR) modulates pathological cardiac hypertrophy through miR-155-mediated IKKi signaling.
TL;DR: CYTOR might play a protective role in cardiac hypertrophy through miR-155 and downstream IKKi and NF-κB signaling, most possibly through serving as a ceRNA for mi R-155 to counteract miR -155-mediated repression of IKBKE.
Journal ArticleDOI
The 5-Lipoxygenase Inhibitor Zileuton Protects Pressure Overload-Induced Cardiac Remodeling via Activating PPARα
Qing-Qing Wu,Wei Deng,Yang Xiao,Jiao-Jiao Chen,Chen Liu,Juan Wang,Yankai Guo,Mingxia Duan,Zhulan Cai,Saiyang Xie,Yuan Yuan,Qi-Zhu Tang +11 more
TL;DR: The data indicated that zileuton protected mice from pressure overload-induced cardiac hypertrophy, fibrosis, and oxidative stress by activating PPARα/NRF2 signaling.
Journal ArticleDOI
Long non-coding RNA Pvt1 modulates the pathological cardiac hypertrophy via miR-196b-mediated OSMR regulation.
TL;DR: In this article, the effect of miR-196b suppression upon the B-raf was shown to be opposite to Pvt1 knockdown, which suggests that miR196b suppressing might significantly attenuate the effects of Pvt 1 knockdown.