J
Julian G. Mercer
Researcher at University of Aberdeen
Publications - 134
Citations - 5575
Julian G. Mercer is an academic researcher from University of Aberdeen. The author has contributed to research in topics: Leptin & Hypothalamus. The author has an hindex of 43, co-authored 133 publications receiving 5259 citations. Previous affiliations of Julian G. Mercer include Rowett Research Institute.
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Journal ArticleDOI
“Eating addiction”, rather than “food addiction”, better captures addictive-like eating behavior
Johannes Hebebrand,Özgür Albayrak,Roger A.H. Adan,Jochen Antel,Carlos Dieguez,Johannes W. de Jong,Gareth Leng,John Menzies,Julian G. Mercer,Michelle Murphy,Geoffrey van der Plasse,Suzanne L. Dickson +11 more
TL;DR: It is concluded that "food addiction" is a misnomer because of the ambiguous connotation of a substance-related phenomenon, and is proposed the term "eating addiction" to underscore the behavioral addiction to eating.
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Hypothalamic thyroid hormone catabolism acts as a gatekeeper for the seasonal control of body weight and reproduction.
Perry Barrett,Francis J. P. Ebling,Sandrine Schuhler,Dana Wilson,Alexander W. Ross,Amy Warner,Preeti H. Jethwa,Anita Boelen,Theo J. Visser,Daniel M. Ozanne,Zoe A. Archer,Julian G. Mercer,Peter J. Morgan +12 more
TL;DR: It is shown that the availability of thyroid hormone within the hypothalamus is a key determinant of seasonal transitions, and a pivotal role for hypothalamic DIO3 and T(3) catabolism in seasonal cycles of body weight and reproduction in mammals.
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Photoperiod regulates arcuate nucleus POMC, AGRP, and leptin receptor mRNA in Siberian hamster hypothalamus
TL;DR: AGRP and NPY mRNAs were extensively coexpressed in the ARC, and their differential regulation by photoperiod and FD is suggestive of transcript-specific regulation at the level of individual neurons.
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Photoperiod differentially regulates the expression of Per1 and ICER in the pars tuberalis and the suprachiasmatic nucleus of the Siberian hamster.
TL;DR: Data demonstrate that photoperiod‐dependent amplitude modulation of Per1 and ICER gene expression in the PT is conserved across species, and reinforce the argument that this phenomenon is driven by melatonin.
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Leptin interacts with glucagon-like peptide-1 neurons to reduce food intake and body weight in rodents
Anthony P. Goldstone,Julian G. Mercer,I. Gunn,Kim M. Moar,C. Mark B. Edwards,M. Rossi,Jane K. Howard,S. Rasheed,Mandy D. Turton,Caroline J. Small,Melanie M. Heath,Donal O'Shea,Joanna Steere,Karim Meeran,M. A. Ghatei,Nigel Hoggard,Stephen R. Bloom +16 more
TL;DR: ICV injection of the specific GLP‐1 receptor antagonist, exendin, at the onset of dark phase, did not affect feeding in saline pre‐treated controls, but blocked the reduction in food intake and body weight of leptin pre‐ treated rats, suggesting that GLp‐1 neurons are a potential target for leptin in its control of feeding.