Julian T. Hoff

TL;DR: The coagulation cascade, haemoglobin breakdown products, and inflammation all play a part in ICH-induced injury and could provide new therapeutic targets and new therapeutic interventions for this severe form of stroke.

TL;DR: Biomechanical alterations likely play a primary role in causing adjacent segment disease, and potential risk factors include instrumentation, fusion length, sagittal malalignment, facet injury, age, and pre-existing degenerative changes.

TL;DR: New knowledge developed under the NCP on the sources, occurrence and pathways of contaminants (organochlorines, Hg, Pb and Cd, PAHs, artificial radionuclides) are highlighted and compelling evidence for close connectivity between the global emission of contaminants from industrial and agricultural activities and the Arctic is provided.

TL;DR: A battery of sensorimotor function tests were used to examine the neurological effects of ICH in the rat and to examine which components of the hematoma are involved in generating those effects.

TL;DR: Limiting hemoglobin degradation coupled with the use of iron chelators may be a novel therapeutic approach to limit brain edema after ICH.