K
Kamejiro Yamashita
Researcher at University of Tsukuba
Publications - 210
Citations - 5019
Kamejiro Yamashita is an academic researcher from University of Tsukuba. The author has contributed to research in topics: Glucagon & Insulin. The author has an hindex of 36, co-authored 210 publications receiving 4929 citations. Previous affiliations of Kamejiro Yamashita include National Cancer Research Institute.
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Bone morphogenetic protein-2 stimulates alkaline phosphatase activity and collagen synthesis in cultured osteoblastic cells, MC3T3-E1.
TL;DR: Results indicate that BMPs act directly on osteoblastic cells and stimulate the expression of the osteoblastics phenotypes.
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Effects of fructo-oligosaccharides on blood glucose and serum lipids in diabetic subjects
TL;DR: In this article, the daily intake of 8.0 g per day of fructo-oligosaccharides for fourteen days was found to significantly reduce mean fasting blood glucose levels by 15 mg/dl, mean serum total cholesterol levels by 19 mg/dL and LDL-cholesterol levels by 17mg/dl in diabetic subjects.
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Endothelin receptor is coupled to phospholipase C via a pertussis toxin-insensitive guanine nucleotide-binding regulatory protein in vascular smooth muscle cells.
Yoh Takuwa,Yoshitoshi Kasuya,Noriko Takuwa,Michiyo Kudo,Masashi Yanagisawa,Katsutoshi Goto,Tomoh Masaki,Kamejiro Yamashita +7 more
TL;DR: Results indicate that the receptor for ET is coupled to phospholipase C via a guanine nucleotide-binding regulatory protein which is distinct from the pertussis toxin substrate in A-10 cells.
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Paraoxonase polymorphism (Gln192-Arg) is associated with coronary heart disease in Japanese noninsulin-dependent diabetes mellitus.
TL;DR: Carriers of the B allele of the Gln192Arg polymorphism in the PONA gene proved to be at increased risk for developing CHD in Japanese NIDDM patients, independent of other known risk factors for CHD, suggesting an important role of the paraoxonase B isoform in the pathogenesis of CHD.
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Comparison of the effects of various C-terminal and N-terminal fragment peptides of glucagon-like peptide-1 on insulin and glucagon release from the isolated perfused rat pancreas
TL;DR: It is concluded that histidine at position 7 of GLP-1 as a free N-terminal amino acid is very important in GLp-1's insulinotropic activity and probably in glucagon-inhibiting activity, and that C- terminal amidation and three C-Terminal amino acids are less important for these activities.