K
Karl S. Matlin
Researcher at University of Chicago
Publications - 73
Citations - 5908
Karl S. Matlin is an academic researcher from University of Chicago. The author has contributed to research in topics: Integrin & Epithelial polarity. The author has an hindex of 33, co-authored 71 publications receiving 5725 citations. Previous affiliations of Karl S. Matlin include University of Basel & Beth Israel Deaconess Medical Center.
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Infectious entry pathway of influenza virus in a canine kidney cell line
TL;DR: The experiments suggest that fowl plague virus enters MDCK cells by endocytosis in coated pits and coated vesicles and is transported to the lysosome where the low pH initiates a fusion reaction ultimately resulting in the transfer of the genome into the cytoplasm.
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Cell fusion by Semliki Forest, influenza, and vesicular stomatitis viruses.
TL;DR: Results show that low-pH-induced fusion is a widespread property of enveloped animal viruses and that it may play a role in the infective process, and suggested a role for the influenza hemagglutinin in the low- pH-dependent membrane fusion activity.
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Reduced temperature prevents transfer of a membrane glycoprotein to the cell surface but does not prevent terminal glycosylation
Karl S. Matlin,Kai Simons +1 more
TL;DR: The transport kinetics of the influenza virus hemagglutinin from its site of synthesis to the apical plasma membrane of Madin-Darby canine kidney cells, a polarized epithelial cell line, were studied by a sensitive tryptic assay, demonstrating that the inhibition at low temperature was reversible.
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Exit of newly synthesized membrane proteins from the trans cisterna of the Golgi complex to the plasma membrane.
TL;DR: The results suggest that the trans cisterna was distinct from the endosome compartment and that the latter was not an obligatory station in the route taken by G protein to the cell surface.
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Pathway of vesicular stomatitis virus entry leading to infection
TL;DR: It is suggested that vesicular stomatitis virus enters MDCK cells by endocytosis in coated pits and coated vesicles, and is transported to the lysosome where the low pH triggers a fusion reaction ultimately leading to the transfer of the genome into the cytoplasm.