Kasey L. Baker

TL;DR: It is shown that adult hippocampal NSCs express vascular endothelial growth factor receptor (VEGFR) 3 and its ligand VEGF-C, which activates quiescent N SCs to enter the cell cycle and generate progenitor cells.

TL;DR: This review will focus on Notch signaling in the vertebrate vascular and nervous systems and examine its role in angiogenesis, neurogenesis, and neurovascular interactions, and highlight the molecular relationships of the Notch pathway with vascular endothelial growth factors and their high-affinity tyrosine kinase VEGF receptors.

TL;DR: Whether neurogenic zones within the MRL brain show enhanced regenerative capacity is examined, and normal formation of chains of migratory neuroblasts along the entire wall and introduction of enhanced green fluorescent protein‐tagged retrovirus into the lateral ventricles confirmed that newly generated neuroblast were able to track into the olfactory bulb.

TL;DR: A novel molecular mechanism involving EphA4 signaling that functions in stem cell niche organization and ultimately neuroblast migration in the anterior forebrain is revealed.

TL;DR: Cx43 is found to be highly expressed by both NSCs and ECs in the SVZ, and its deletion in either cell type leads to increased NSC proliferation and neuroblast generation, suggesting that Cx43-mediated NSC-EC interactions maintain NSC quiescence.