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Katherine P. Rankin

Researcher at University of California, San Francisco

Publications -  212
Citations -  21238

Katherine P. Rankin is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Frontotemporal dementia & Dementia. The author has an hindex of 54, co-authored 169 publications receiving 16202 citations. Previous affiliations of Katherine P. Rankin include National and Kapodistrian University of Athens & University of Western Ontario.

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Atrophy progression in semantic dementia with asymmetric temporal involvement: A tensor-based morphometry study

TL;DR: The anatomic substrates of the previously reported clinical evolution of LTLV and RTLV are identified into a unique 'merged' clinical syndrome characterized by semantic and behavioral deficits and bilateral temporal atrophy.
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Detecting Sarcasm from Paralinguistic Cues: Anatomic and Cognitive Correlates in Neurodegenerative Disease

TL;DR: This paper investigated the neuroanatomy underlying failure to understand sarcasm from dynamic vocal and facial paralinguistic cues and found that sarcasm comprehension was predicted by smaller volume in bilateral posterior parahippocampi (PHc), temporal poles, and R medial frontal pole (pFWE < 0.05).
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Clinicopathological correlations in behavioural variant frontotemporal dementia.

TL;DR: In this paper, the authors explored clinicopathological correlations in a large bvFTD cohort and used a combination of known predictive factors (genetic mutations, motor features, or striking atrophy patterns) and the results of a discriminant function analysis that incorporated clinical, neuroimaging and neuropsychological data.
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NIH EXAMINER: conceptualization and development of an executive function battery.

TL;DR: This effort to develop psychometrically robust executive measurement tools that would be accepted by the neurology clinical trials and clinical research communities resulted in a series of tasks targeting working memory, inhibition, set shifting, fluency, insight, planning, social cognition and behavior.
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The salience network causally influences default mode network activity during moral reasoning

TL;DR: These findings link recent work on the dynamic interrelationships between large-scale brain networks to observable impairments in dementia syndromes, which may shed light on how diseases that target one network also alter the function of interrelated networks.