K
Katsutoshi Goto
Researcher at University of Tsukuba
Publications - 231
Citations - 31742
Katsutoshi Goto is an academic researcher from University of Tsukuba. The author has contributed to research in topics: Endothelin 1 & Endothelin receptor. The author has an hindex of 63, co-authored 228 publications receiving 31015 citations. Previous affiliations of Katsutoshi Goto include University of Texas Southwestern Medical Center.
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Relationship between serum glufosinate level and development of serious poisoning caused by the ingestion of Basta®, a herbicide containing glufosinate
Kanji Koyama,Yasuo Hirose,Takanori Okuda,Akiko Motokawa,Noriyoshi Ohashi,Misa Iwai,Naoki Matsumiya,Yukio Ono,Yoshitaka Ishii,Takayuki Matsumoto,Shuuya Kusumoto,Masaki Kawano,Masayuki Suzukawa,Kaichiro Tanba,Hiroyuki Kobayashi,Masaaki Nagai,Hideko Yamabe,Hisashi Akuzawa,Katsutoshi Goto +18 more
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Influence of acidosis on cardiotonic effects of milrinone
TL;DR: The results indicate that the inotropic effect of milrinone is attenuated by acidosis due, at least in part, to decreased cAMP formation in acidotic muscle.
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Exercise-induced tissue-specific change in gene expression of endothelin-1.
TL;DR: It is demonstrated that acute exercise causes tissue-specific changes in the production of ET-1 and that these alterations may participate in the integrated physiological response during exercise.
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Pharmacological studies of imidazoline derivatives. I. A comparison of the effect on pre- and postsynaptic alpha-adrenoceptors in the rat vas deferens.
TL;DR: The effects of 8 imidazoline derivatives on pre- and postsynaptic alpha-adrenoceptors and [3H]norepinephrine release were investigated in the rat vas deferens and were compared with those of reference drugs, yohimbine, phentolamine, tolazoline, and prazosin.
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Time course alteration of endothelin-1 gene expression in the heart during exercise and recovery from post-exercise periods in rats.
TL;DR: It is suggested that a bout of exercise causes time-related enhancement of gene expression of ET-1 in the rat hearts during acute exercise and 24 hour post-exercise periods, which may participate in mechanisms of exercise-induced and/or training-induced adaptive responses of the heart.