K
Kenji Kosaka
Researcher at Yokohama City University
Publications - 274
Citations - 20373
Kenji Kosaka is an academic researcher from Yokohama City University. The author has contributed to research in topics: Dementia with Lewy bodies & Dementia. The author has an hindex of 51, co-authored 274 publications receiving 18520 citations. Previous affiliations of Kenji Kosaka include Hochschule Hannover & Max Planck Society.
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Journal Article
[A clinicopathological study of Pick's disease--our sixty autopsied cases (author's transl)].
Journal Article
[Some problems on the clinical phenotype of Machado-Joseph disease in relation between their ages at onset].
Iwabuchi K,Kogure T,Tatsuro Oda,Kato Y,Ohtani K,Endo K,Kenji Kosaka,Naoji Amano,Saburo Yagishita +8 more
TL;DR: It is proposed that three phenotypes of Machado-Joseph disease (MJD) are closely related to the patients' ages at onset and clinical progression of the disease.
Journal Article
[Pick's disease with concomitant traumatic brain damage--five autopsied cases and a critical review of literatures (author's transl)].
Journal ArticleDOI
An autopsied case of corticobasal degeneration showing severe cerebral atrophy over a protracted disease course of 16 years.
Daizo Kondo,Daizo Kondo,Hiroaki Hino,Katsuhiko Shibuya,Koshiro Fujisawa,Kenji Kosaka,Yoshio Hirayasu,Ryoko Yamamoto,Koji Kasanuki,Michiko Minegishi,Kiyoshi Sato,Masato Hosokawa,Tetsuaki Arai,Heii Arai,Eizo Iseki +14 more
TL;DR: The patient was a 72‐year‐old Japanese woman who started having difficulty performing daily work and developed agraphia, and was pathologically diagnosed as having corticobasal degeneration, which likely accounts for the severe white matter degeneration and accumulation of 3R tau in NFTs.
Journal ArticleDOI
Frequency and distribution of TUNEL‐positive neurons in brains of dementia with Lewy bodies: Comparison with those in brains of Alzheimer's disease
TL;DR: It is suggested that neuronal damage showing DNA fragmentations occurs in DLB brains as well as in AD and DS brains, and that it is accelerated by progression of Lewy pathology as wellAs Alzheimer pathology, although it is not directly related to their pathological hallmarks.